A cool case for #cardiotwitter #neurotwitter -- wondering if others have seen this happen!

A 70 yo W with history of HTN presented with significant IVH from a ruptured AVM.

Admission EKG showed this:
A #brugada pattern. She had no personal or family history of syncope / sudden death. And on admission (time of this EKG) she was not febrile. About 12 hours later we repeated the EKG:
Trops normal and ECHO later in the day demonstrated a normal EF and grade 1 DD, but no wall motion abnormality. No apical ballooning. There was mildly increase LV wall thickness.
Checked another EKG the next AM. Looks about the same.

There were no arrhythmias on tele. She continued to be afebrile.
A few days later (about 6 days out from IVH), she developed a pneumonia and spiked a temperature.

Repeat EKG demonstrated:
Absent Brugada pattern, despite the fever (1st of her admission).

So, #cardiotwitter, do we just chalk this up to a brain-heart ❤️🧠 connection thing? The stress of IVH? Inflammation? Never personally seen this pattern with neuro patients so I'm curious!
And -- Is it likely to return? Or like all the QTc prolongations & other transient arrhythmias we find is this unlikely to reoccur.

Thoughts on why this pattern might be found with neuro injury?

@PPodrid @sgleeMD @guroledip @emoryheart @AaronLBerkowitz @Capt_Ammonia

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More from @caseyalbin

28 May
1/
1st week of NeuroICU fellowship. A #tweetorial summary:

1⃣ Pt in DI. Give anti-diuretic hormone (ADH), call it “pit drip”
2⃣Pt in distributive shock. Give ADH, call it “vaso”
3⃣Pt on ASA needs EVD. Give ADH (sort of), call it “DDAVP”
4⃣ Fellow postcall & confused, give…. Image
2/
Just kidding… everyone knows the drug for that is Image
3/
All the names and purposes of ADH had me feeling ⬇️

So – a review of all things ADH including:
✅It’s various aliases
✅Receptors and function
✅Clinical utility in NeuroICU (+general ICUs)
Read 25 tweets
12 May
1/
What is the most worthless electrolyte on the BMP, and why is it chloride?

Agree?
A #tweetorial 🧵 to change your mind…

And if you’re thinking, why in the world is this a #neurotweetorial? Read on. #neurotwitter @MedTweetorials
2/
First, this #tweetorial is based on a lecture given in @emoryneurocrit didactics by one of our *awesome* teachers: Dr. Ofer Sadan (@neuro_intensive), and is shared in #tweetorial format w/ his permission.
3/
So, again, why is a #neurologist interested in chloride?

Because neurologists ❤️ giving chloride.

Ur thinking, “No. I have literally never ordered chloride.” … But, think of all the 23.4% and 3% sodium you have ordered.

All that sodium comes with a bystander: Chloride.
Read 20 tweets
29 Apr
A #tweetorial #medtweetorial
@MedTweetorials

1/
3 AM: Pager Pager Pager

Me [very groggily]: Hey, what’s up?

Awesome overnight APP: Sorry to wake you! But, Mrs. Very-Small-Stroke-NIHSS-2-Had-Been-Clinically-Stable-All-Day just PEA arrested.

Me [not so groggily]:
2/
Awesome APP: We successfully got ROSC (bc team
@emoryneurocrit
= Flexed bicepsStar-struck) and have him stabilized. Going to scan now!

So, #neurotwitter, where’s the lesion?
3/
Trick question.

All of these areas through neurologic pathology – stroke/seizure/bleed - could all have caused a sudden death (or at the very least sudden LOC).
Read 24 tweets
31 Mar
1/
Attending: “But never anticoagulation for an intracranial dissection!”

Me as a resident: “Of course. Totally!”

Also me ⬇️: [googling “where do the internal carotid arteries and vertebral arteries become intracranial?”]

Feel familiar? A #tweetorial @MedTweetorials
2/
With the TREAT-CAD trial, lots of talk about dissection treatment. Whether your team anti-platelets or team anticoagulation (🙋🏻 Must. Give. Heparin (@MGHNeurology) 4 ever. I know you feel this, @namorris!) consideration about the location of dissection is possibly important.
3/
Also, regardless of your team… TREAT-CAD was not able to demonstrate non-inferiority of ASA, just saying.

Aspirin versus anticoagulation in cervical artery dissection (TREAT-CAD): an open-label, randomised, non-inferiorit… pubmed.ncbi.nlm.nih.gov/33765420/
Read 12 tweets
26 Mar
1/
Your patient that is “over breathing” the vent may still be dead by brain criteria.

A #tweetorial #medtweetorial for providers in #criticalcare #EM #neuro #neurocriticalcare. @MedTweetorials
2/
First and foremost, let’s be clear that to be dead by brain criteria, the patient must have cessation of ALL brain function *INCLUDING absence of respiratory drive.*

Thinking "But... I thought you just said...."?
3/
The contradiction here lies in that ventilators are sometimes too sensitive.
Read 13 tweets
18 Feb
1/ Alright, #Neurotwitter, the votes for today’s #neuroDDxThursday were overwhelmingly in favor of multiple cranial neuropathies!

Thought about one slide, but this needs a #tweetorial! So that you aren't 😬😬😬 when confronted with this:

#MedEd #FOAMed @MedTweetorials
2/
Reminder: The 12 cranial nerve nuclei are located in the brainstem, and if you have trouble remembering where they are, welcome to the club. Here’s a reminder! Will post the medulla section Monday, stay tuned.
3/
We’ll move from central to peripheral etiologies.

The brainstem is like Times Square in NYC- so much going on in a very small space.

A small insult can easily cause damage to multiple cranial nerves. amiright, #stroketwitter?
Read 15 tweets

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