➡️Early Rep is a common cause of STE. Typically seen in younger males and in athletic.
➡️It's a risk factor for sudden cardiac death, as it increases the risk for idiopathic ventricular fibrillation.
➡️Typically produces a notched J-point with STE that is <3 mm on the ECG
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➡️ECG features of Pericarditis:
👉Widespread saddle shaped (concave upward) STE
👉TWI only after the ST have returned to baseline
👉Q waves do not develop.
👉PR segment depression
👉ECG Changes occur over the course of several weeks, in contrast to rapid evolution in STEMI
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➡️ ECG features that favor STEMI over Pericarditis:
👉 Reciprocal ST-segment depression or T-wave inversions
👉 ST Elevation that is flat or horizontal should be considered an STEMI until proven otherwise.
👉 Rapid evolution of ST segment changes
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➡️ In the setting of LBBB, the ST segment is directly opposite to that of the main QRS complex vector.
➡️ In leads V1 to V3, the QRS vector is negative, thereby producing ST Elevation.
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➡️ LVH produces discordant ST Elevation, with a deep S wave in leads V1 to V3 and a tall R wave in leads I, aVL, V5, and V6.
➡️ Left ventricular aneurysm may have STE along with deep anterior or septal Q waves in leads V1 to V3. Do not cause dynamic ECG abnormalities.
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➡️ Hyperkalemia is often referred to as the “great imitator” of ECG abnormalities. Can produce numerous ECG findings that can easily be misdiagnosed.
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➡️ Prinzmetal Angina and Takotsubo Cardiomyopathy are often diagnosed as STEMI. These diagnoses are often made following emergent cardiac catheterization.
➡️ Acids cause coagulative necrosis, which results in a self-limiting burn pattern
➡️ Alkaline materials induce liquefactive necrosis with diffusion into deeper layers of the injured mucosa. Even low concentrations of alkaline ingestion can cause extensive injury.
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➡️ Caustic Ingestion can provoke injury from the mouth, the airway, down through the esophagus to the small intestine.
➡️ Long-term complications can lead to strictures and an increased risk of esophageal cancer.
➡️ Boerhaave syndrome is a spontaneous rupture of the esophagus from barotrauma related to retching or any sudden increase in intraabdominal pressure against a closed glottis.
➡️ Boerhaave syndrome should be suspected in any patient who presents with retrosternal chest pain, neck pain, or epigastric pain, especially if it began after an episode of severe retching or bearing down against a closed glottis (weight lifting, defecation, childbirth, etc)
➡️ Additional critical diagnoses to consider that can be detected with ECG:
👉Ventricular Preexcitation (WPW)
👉Brugada syndrome
👉Long or short QT syndromes
👉Hypertrophic Cardiomyopathy
👉Arrhythmogenic right ventricular dysplasia
👉Catecholaminergic polymorphic VT
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➡️ Ventricular Preexcitation (WPW)
👉 PR segment < 120 ms
👉 QRS complex > 110 ms
👉 Slurred upstroke of the initial part of the R wave (delta wave)
➖ Type A: delta wave in all precordial leads, R > S in lead V1
➖ Type B: negative delta waves in leads V1 and V2
➡️ Diuretics may not be the preferred initial therapy for those patients with vascular failure, who are often euvolemic, or those with cardiogenic shock, who are often hypovolemic.
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➡️ In these patients, the emergency provider should first optimize preload and afterload reduction with the use of noninvasive positive pressure ventilation (NPPV), nitrates, or inotropes as indicated.