➡️ Additional critical diagnoses to consider that can be detected with ECG:
👉Ventricular Preexcitation (WPW)
👉Brugada syndrome
👉Long or short QT syndromes
👉Hypertrophic Cardiomyopathy
👉Arrhythmogenic right ventricular dysplasia
👉Catecholaminergic polymorphic VT
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➡️ Ventricular Preexcitation (WPW)
👉 PR segment < 120 ms
👉 QRS complex > 110 ms
👉 Slurred upstroke of the initial part of the R wave (delta wave)
➖ Type A: delta wave in all precordial leads, R > S in lead V1
➖ Type B: negative delta waves in leads V1 and V2
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➡️ Brugada Syndrome
👉Type 1
➖Coved ST elevation
➖>2 mm of ST elevation in 2 or more precordial leads (V1 to V3) and negative T wave
👉Type 2
➖Saddle back ST elevation
➖>2 mm of ST elevation in 2 or more precordial leads
👉Type 3
➖Either type <2 mm ST elevation
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➡️ Long QT Syndrome
👉 Prolonged if QTc > 440 ms in men or >460 ms in females
👉 Increased risk of dysrhythmias when the QTc > 500 ms
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➡️ Short QT Syndrome
👉 Shortened if QTc < 330 ms in males or <340 ms in females
👉 Short, or absent, ST-segment with peaked appearance of the T wave
👉 Etiologies to consider include congenital shortening, digoxin toxicity, and hypercalcemia.
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➡️ Hypertrophic cardiomyopathy
👉 Deep Q waves in the lateral (I, aVL, V5-V6) and inferior (II, III, aVF) leads
👉 Left ventricular hypertrophy (LVH)
👉 Left atrial enlargement
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➡️ Arrhythmogenic Right Ventricular Dysplasia (ARVD)
👉 Epsilon wave (small positive deflection at end of QRS complex)
👉 T-wave inversions in V1 to V3
👉 Prolonged QRS in V1 to V3 (100 to 120 ms)
👉 Slurred S-wave upstroke in V1 to V3 (50 to 55 ms)
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➡️ Catecholaminergic Polymorphic VT
👉 ECG is typically normal at rest.
👉 Exercise classically induces VT from adrenergic activation.
👉 Bidirectional tachycardia
👉 Ventricular ectopy is often noted when the heart rate increases above 100 beats per minute.
➡️ Acids cause coagulative necrosis, which results in a self-limiting burn pattern
➡️ Alkaline materials induce liquefactive necrosis with diffusion into deeper layers of the injured mucosa. Even low concentrations of alkaline ingestion can cause extensive injury.
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➡️ Caustic Ingestion can provoke injury from the mouth, the airway, down through the esophagus to the small intestine.
➡️ Long-term complications can lead to strictures and an increased risk of esophageal cancer.
➡️ Boerhaave syndrome is a spontaneous rupture of the esophagus from barotrauma related to retching or any sudden increase in intraabdominal pressure against a closed glottis.
➡️ Boerhaave syndrome should be suspected in any patient who presents with retrosternal chest pain, neck pain, or epigastric pain, especially if it began after an episode of severe retching or bearing down against a closed glottis (weight lifting, defecation, childbirth, etc)