2/Everyone needs some support. Just like the tongue is supported by the hyoid, the main laryngeal support is the cricoid cartilage. It's like your favorite chair, with all the remotes next to it--not only does the cricoid provide support, it also connects structures in the larynx
3/ Although not official, the cricoid is like the real first ring of the trachea. Except it has an odd signet ring shape. The bulbous back part that makes it look like a signet ring provides the important articulation with the arytenoids
4/Arytenoid cartilages are these funny elf-hat shaped cartilages that are attached to the vocal cords. When the arytenoids move, your vocal cords move, and that's how phonation is created.
5/Arytenoids move by muscles that are stretched out between them, in a criss-cross pattern like shoe laces (transverse/oblique arytenoid muscles). When these muscles contract, it is like tightening shoe laces, which pull and rotate the arytenoids together
6/When the shoe laces are tightened or loosened, the arytenoids swing open or closed like an old fashioned saloon door--and open and close the vocal cords with them!
7/How the cricoid & arytenoids look on imaging depends on the level. Higher up, you are at the articulation of the arytenoids w/the signet ring & you see the crico-arytenoid joint. Lower down, you only see the posterior signet ring, & only at the bottom is entire ring visible
8/Larynx is so important to breathing & phonation that it needs protection--that is the thyroid cartilage. Thyroid cartilage is like a shield for the larynx. But it's huge, like those old Roman shields covering almost the entire body--that's the thyroid cartilage for the larynx
9/Thyroid cartilage is shaped like a bad tiara--except it has points going both up & down. It looks like one of those tiaras you thought would look great w/your prom dress at 16 &now you are stuck w/horrible photos where you look like a bad Disney princess--or was that just me?
10/Because it is open posteriorly, like a tiara, and slightly pointed, on imaging--in cross section--it looks like a upside down letter V
11/So here's the summary of laryngeal supporting structures & how they relate to other structures. Hopefully this has shined some light on the black box of voice box anatomy!
Laryngeal anatomy is too complex for one tweetorial--this is just the beginning--stay tuned for more 😉
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2/A key concept in these fxs is dental occlusion. The jaw is meant to chew. To crush food, teeth need to come as close together as possible—occlusion. Each tooth needs to meet up with its counterpart that fits with it, so no room is left for food—and food will be crushed.
3/Occlusion can be lost w/a fx. The importance of dental occlusion makes mandibular fxs different from other fxs. Usually, we want to fix a bone so that it lines up again. But for the mandible, we want to fix it so the TEETH line up again—so chewing will work.
2/ With CSF leaks, everyone knows about brain sagging. But this can happen w/other pathologies, ie Chiari 1. Other findings can be seen on brain MRI in CSF leaks. But what are these findings? Are some findings more suggestive than others? Do more findings = ⬆️suspicion?
3/Dobrocky et al. looked at 9 quantitative & 7 qualitative signs seen on brain MRI in CSF leaks to see which are most important. Depending on type & # of findings, they developed a score to indicate what level of suspicion you should have for a leak. pubmed.ncbi.nlm.nih.gov/30776059/
2/ fMRI is based on a principle called “neurovascular coupling.” This is the principle if there is increased neuronal activity in a region, there will be increased blood flow to that region to meet the increased demand
3/ Think of it like a baby crying because it is hungry—parents immediately rush to feed it. The increased oxygen demand of the neurons immediately brings increased fuel to feed it.
1/ “You don’t get points for having your needle in the right place if you don’t get a diagnosis.” When we biopsy the skullbase we work to get a diagnosis.
A sort of #tweetorial but more like a 🧵about our skullbase biopsy system. #FOAMed#medtwitter#neurosurgery#neurotwitter
2/ Unless the lesion is difficult to diagnosis w/FNA (ie, schwannoma), we begin by FNA w/an 18g draw needle & a 22g Quincke needle. We do not aspirate, b/c the skullbase is very vascular, & too much blood will be drawn up, making it difficult to tell if the sample is diagnostic.
3/ However, if we are not getting a diagnosis with FNA, we will move to a core. If it is a deep lesion, we will use the Biopince system, beginning with a 17g, 7 cm introducer. This is an example of IgG4 disease of the trigeminal nerve that failed FNA and required a core
1/
Why is cranial nerve 6 uniquely affected by⬆️ intracranial pressure? Why is it special? A common question after the CN6 tweetorial.
Here is a maybe #tweetorial, but maybe a🧵about why CN6 is alone affected by ⬆️ pressure. #FOAMed#medtwitter#Medstudenttwitter#neurotwitter
2/ Think of the intracranial CSF space like a balloon, distended by CSF instead of air. Cranial nerves begin inside the balloon, and then they exit as they begin their extradural portion
3/ Most cranial nerves move immediately away from the CSF space after they exit—usually going out through their respective foramina. However, CN6 uniquely runs along the outside of the “balloon” in Dorello canal
1/ It is rare a patient tells you where their pathology is—but they do when they tell you they have a cranial nerve palsy—you just have to know where to look!
A #tweetorial about the Six Syndromes of the Sixth Cranial Nerve. #FOAMed#medtwitter#Medstudenttwitter@medtweetorials
2/ To understand the six syndromes, you have to know the anatomy of the 6th nerve. It starts in the brainstem, at the facial colliculus—what looks like the butt of the brainstem. It then travels anteriorly through the brainstem to exit out the ventral surface of the pons.
3/ It exits the brainstem at the inferior aspect of the pons—under what looks like pot belly of the pons and then travels anteriorly in the subarachnoid space towards the clivus