2/Some believe that the peaked, mountain like appearance of the frontal lobes is a critical sign of a life-threatening complication & should be called & reported. Others believe it is too non-specific, is commonly seen when there isn’t tension & should be retired. Who’s right?
3/First, let’s clarify about what the Mt Fuji sign actually is. Most are familiar with the fact that large collections of pneumocephalus can compress the frontal lobes—making them look like the slopes of a mountain. But this isn’t actually enough to call Mt Fuji.
4/You also need to see frontal lobe separation. This means the subdural air tension is greater than CSF surface tension between the frontal lobes--one of the highest liquid surface tensions—so you know pressure is high. This little V is why it looks like Mt Fuji, not any mountain
5/Why do we get tension pneumocephalus? 3 main ways. (1) Upside down coke bottle effect w/a CSF leak. As liquid drips out w/a CSF leak, nature abhors a vacuum, so air rushes in to replace it. If outside pressure is higher than CSF pressure, more air will come in & create tension
6/Here’s a skullbase CSF leak creating pneumocephalus. As CSF leaks out, air replaces it. If air pressure is higher than intracranial pressure, more air will come in. The worst tension pneumocephalus I ever saw was a pt w/an unknown sphenoid sinus skullbase leak they put on CPAP!
7/Next mechanism is the ball valve mechanism. Air gets in through a defect (from trauma, surgery, etc). Increased pressure eventually pushes down on the brain, causing the brain to close the defect so the air can’t escape. This is the same mechanism seen w/tension pneumothorax.
8/Final mechanism is use of nitrous oxide in neuroanesthesia. If the pt has a subdural collection (usually w/air in the operative setting), nitrous oxide enters the subdural 34 times faster than it diffuses out as nitrogen into the blood stream—creating increased pressure/tension
9/This is why nitrous oxide is no longer commonly used in neuroanesthesia. Decreased use of nitrous oxide is also why tension pneumocephalus is less common in the post-operative setting now than it was in the past.
10/So how helpful is the Mt Fuji sign for determining tension pneumocephalus post-operatively (a time when pneumocephalus is common)? Well it turns out, the sign can be seen in 1/3 pts without a neurosurgical emergency. So it is not very specific and can cause overcalling
11/More importantly is how the pt is doing clinically. Significant pneumocephalus can be seen post op—but if the pt is not declining, even large amounts of pneumocephalus can be managed by putting the pt on 100% O2--just like w/a pneumothorax--w/good results.
12/So remember it isn’t just one sign—it is the whole picture of how the pt is doing clinically. Don’t lose sight of the forest for the mountain!
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Brain MRI anatomy is best understood in terms of both form & function.
Here’s a short thread to help you to remember important functional brain anatomy--so you truly can clinically correlate!
2/Let’s start at the top. At the vertex is the superior frontal gyrus. This is easy to remember, bc it’s at the top—and being at the top is superior. It’s like the superior king at the top of the vertex.
3/It is also easy to recognize on imaging. It looks like a big thumb pointing straight up out of the brain. I always look for that thumbs up when I am looking for the superior frontal gyrus (SFG)
@TheAJNR 2/Everyone knows about the spot sign for intracranial hemorrhage
It’s when arterial contrast is seen within a hematoma on CTA, indicating active
extravasation of contrast into the hematoma.
But what if you want to know before the CTA?
@TheAJNR 3/Turns out there are non-contrast head CT signs that a hematoma may expand that perform similarly to the spot sign—and together can be very accurate.
1/My hardest thread yet! Are you up for the challenge?
How stroke perfusion imaging works!
Ever wonder why it’s Tmax & not Tmin?
Do you not question & let RAPID read the perfusion for you? Not anymore!
2/Perfusion imaging is based on one principle: When you inject CT or MR intravenous contrast, the contrast flows w/blood & so contrast can be a surrogate marker for blood.
This is key, b/c we can track contrast—it changes CT density or MR signal so we can see where it goes.
3/So if we can track how contrast gets to the tissue (by changes in CT density or MR signal), then we can approximate how BLOOD is getting to the tissue.
And how much blood is getting to the tissue is what perfusion imaging is all about.