2/Some believe that the peaked, mountain like appearance of the frontal lobes is a critical sign of a life-threatening complication & should be called & reported. Others believe it is too non-specific, is commonly seen when there isn’t tension & should be retired. Who’s right?
3/First, let’s clarify about what the Mt Fuji sign actually is. Most are familiar with the fact that large collections of pneumocephalus can compress the frontal lobes—making them look like the slopes of a mountain. But this isn’t actually enough to call Mt Fuji.
4/You also need to see frontal lobe separation. This means the subdural air tension is greater than CSF surface tension between the frontal lobes--one of the highest liquid surface tensions—so you know pressure is high. This little V is why it looks like Mt Fuji, not any mountain
5/Why do we get tension pneumocephalus? 3 main ways. (1) Upside down coke bottle effect w/a CSF leak. As liquid drips out w/a CSF leak, nature abhors a vacuum, so air rushes in to replace it. If outside pressure is higher than CSF pressure, more air will come in & create tension
6/Here’s a skullbase CSF leak creating pneumocephalus. As CSF leaks out, air replaces it. If air pressure is higher than intracranial pressure, more air will come in. The worst tension pneumocephalus I ever saw was a pt w/an unknown sphenoid sinus skullbase leak they put on CPAP!
7/Next mechanism is the ball valve mechanism. Air gets in through a defect (from trauma, surgery, etc). Increased pressure eventually pushes down on the brain, causing the brain to close the defect so the air can’t escape. This is the same mechanism seen w/tension pneumothorax.
8/Final mechanism is use of nitrous oxide in neuroanesthesia. If the pt has a subdural collection (usually w/air in the operative setting), nitrous oxide enters the subdural 34 times faster than it diffuses out as nitrogen into the blood stream—creating increased pressure/tension
9/This is why nitrous oxide is no longer commonly used in neuroanesthesia. Decreased use of nitrous oxide is also why tension pneumocephalus is less common in the post-operative setting now than it was in the past.
10/So how helpful is the Mt Fuji sign for determining tension pneumocephalus post-operatively (a time when pneumocephalus is common)? Well it turns out, the sign can be seen in 1/3 pts without a neurosurgical emergency. So it is not very specific and can cause overcalling
11/More importantly is how the pt is doing clinically. Significant pneumocephalus can be seen post op—but if the pt is not declining, even large amounts of pneumocephalus can be managed by putting the pt on 100% O2--just like w/a pneumothorax--w/good results.
12/So remember it isn’t just one sign—it is the whole picture of how the pt is doing clinically. Don’t lose sight of the forest for the mountain!
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Circle of Willis anatomy is king, while the vascular anatomy of the blood supply to the dura is the poor, wicked step child of vascular anatomy that is often forgotten
@TheAJNR 3/But dural vascular anatomy & supply are important, especially now that MMA embolizations are commonly for chronic recurrent subdurals.
It is also important for dural arteriovenous fistulas.
1/I always say you can tell a bad read on a spine MR if it doesn’t talk about lateral recesses.
What will I think when I see your read? Do you rate lateral recess stenosis?
Here’s a thread on lateral recess anatomy & a grading system for lateral recess stenosis
2/First anatomy.
Thecal sac is like a highway, carrying the nerve roots down the lumbar spine.
Lateral recess is part of the lateral lumbar canal, which is essentially the exit for spinal nerve roots to get off the thecal sac highway & head out into the rest of the body
3/Exits have 3 main parts.
First is the deceleration lane, where the car slows down as it starts the process of exiting.
Then there is the off ramp itself, and this leads into the service road which takes the car to the roads that it needs to get to its destination
1/Do radiologists sound like they are speaking a different language when they talk about MRI?
T1 shortening what? T2 prolongation who?
Here’s a translation w/an introductory thread to MRI.
2/Let’s start w/T1—it is #1 after all! T1 is for anatomy
Since it’s anatomic, brain structures will reflect the same color as real life
So gray matter is gray on T1 & white matter is white on T1
So if you see an image where gray is gray & white is white—you know it’s a T1
3/T1 is also for contrast
Contrast material helps us to see masses
Contrast can’t get into normal brain & spine bc of the blood brain barrier—but masses don’t have a blood brain barrier, so when you give contrast, masses will take it up & light up, making them easier to see.