Lea Alhilali, MD Profile picture
Sep 8, 2022 12 tweets 7 min read Read on X
1/Controversy in radiology gets tense! The Mt Fuji sign for tension pneumocephalus is under scrutiny.When should you call it?
A #tweetorial about #imaging this important #neurosurgery complication
#medtwitter #FOAMed #FOAMrad #medstudent #neurorad #radres #meded #neurotwitter Image
2/Some believe that the peaked, mountain like appearance of the frontal lobes is a critical sign of a life-threatening complication & should be called & reported. Others believe it is too non-specific, is commonly seen when there isn’t tension & should be retired. Who’s right? Image
3/First, let’s clarify about what the Mt Fuji sign actually is. Most are familiar with the fact that large collections of pneumocephalus can compress the frontal lobes—making them look like the slopes of a mountain. But this isn’t actually enough to call Mt Fuji. Image
4/You also need to see frontal lobe separation. This means the subdural air tension is greater than CSF surface tension between the frontal lobes--one of the highest liquid surface tensions—so you know pressure is high. This little V is why it looks like Mt Fuji, not any mountain Image
5/Why do we get tension pneumocephalus? 3 main ways. (1) Upside down coke bottle effect w/a CSF leak. As liquid drips out w/a CSF leak, nature abhors a vacuum, so air rushes in to replace it. If outside pressure is higher than CSF pressure, more air will come in & create tension Image
6/Here’s a skullbase CSF leak creating pneumocephalus. As CSF leaks out, air replaces it. If air pressure is higher than intracranial pressure, more air will come in. The worst tension pneumocephalus I ever saw was a pt w/an unknown sphenoid sinus skullbase leak they put on CPAP! Image
7/Next mechanism is the ball valve mechanism. Air gets in through a defect (from trauma, surgery, etc). Increased pressure eventually pushes down on the brain, causing the brain to close the defect so the air can’t escape. This is the same mechanism seen w/tension pneumothorax. Image
8/Final mechanism is use of nitrous oxide in neuroanesthesia. If the pt has a subdural collection (usually w/air in the operative setting), nitrous oxide enters the subdural 34 times faster than it diffuses out as nitrogen into the blood stream—creating increased pressure/tension Image
9/This is why nitrous oxide is no longer commonly used in neuroanesthesia. Decreased use of nitrous oxide is also why tension pneumocephalus is less common in the post-operative setting now than it was in the past. Image
10/So how helpful is the Mt Fuji sign for determining tension pneumocephalus post-operatively (a time when pneumocephalus is common)? Well it turns out, the sign can be seen in 1/3 pts without a neurosurgical emergency. So it is not very specific and can cause overcalling Image
11/More importantly is how the pt is doing clinically. Significant pneumocephalus can be seen post op—but if the pt is not declining, even large amounts of pneumocephalus can be managed by putting the pt on 100% O2--just like w/a pneumothorax--w/good results. Image
12/So remember it isn’t just one sign—it is the whole picture of how the pt is doing clinically. Don’t lose sight of the forest for the mountain!

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More from @teachplaygrub

Aug 1
1/They say form follows function!

Brain MRI anatomy is best understood in terms of both form & function.

Here’s a short thread to help you to remember important functional brain anatomy--so you truly can clinically correlate! Image
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Do you know when a hematoma is going to expand?

Read on for month’s @theAJNR SCANtastic on all you need to know about imaging intracranial hemorrhage!

ajnr.org/content/46/7/1…Image
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But what if you want to know before the CTA? Image
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How can you remember what they are? Image
Read 9 tweets
Jul 25
1/Time to go with the flow!

Hoping no one notices you don’t know the anatomy of internal carotid (ICA)?

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Here’s a thread to help you siphon off some information about ICA anatomy! Image
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First floor is skullbase/carotid canal. Next it stops at the cavernous sinus, before finally reaching the rooftop balcony of the intradural space.Image
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C1 is in the lobby or neck.

You can remember this b/c the number 1 looks elongated & straight like a neck. Image
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Jul 23
1/My hardest thread yet! Are you up for the challenge?

How stroke perfusion imaging works!

Ever wonder why it’s Tmax & not Tmin?

Do you not question & let RAPID read the perfusion for you? Not anymore! Image
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And how much blood is getting to the tissue is what perfusion imaging is all about. Image
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1/Do you know all the aspects of, well, ASPECTS?

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Do you know pc-ASPECTS?!

Here’s how to remember pc-ASPECTS! Image
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If you need a review: here’s my thread on ASPECTS: Image
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Even w/recanalization, many of these pts do poorly bc of the extent of already infarcted tissue.

So there’s a need to quantitate the amount of infarcted tissue in these ptsImage
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Does seeing an infarct in the medulla cause your heart to skip a beat?

Does medullary anatomy send you into respiratory arrest?

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2/The medulla is like a toll road.

Everything going down into the cord must pass through the medulla & everything from the cord going back up to the brain must too.

That’s a lot of tracts for a very small territory. Luckily you don’t need to know every tract Image
3/Medulla has 4 main vascular territories, spread out like a fan: anteromedial, anterolateral, lateral, and posterior.

You don’t need to remember their names, just the territory they cover—and I’ll show you how Image
Read 18 tweets

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