Depleted Lymphocyte B & T cells from persistent SARS-CoV viral infection of renal epithelium cells will no doubt lead to acquire all kinds of immunity dysfunction and will appear as CLL.
Patients are being tested for digestive disorders and diagnosed with CLL months after a SARS infection that nobody is testing for or tracking - because attempting to detect the viral persistence is invasive procedure.
What I really like about this study from just before the pandemic is use the term “repeated insults”!
Are they discussing lymphopenia draining naïve t-cells during graft procedure? YES! Guess what persistent renal infection would look like! Donated liver! ncbi.nlm.nih.gov/pmc/articles/P…
The astounding rise in #CLL diagnosis across the board is absolutely INSULTING!
But at least they are testing for t-cell depletion?
Because nobody was testing for Hodgkin Lymphoma BEFORE, suddenly patients are being blamed for “life choices” that must have exasperated EXISTING #AirborneAIDS!
We are slowly finding out SARS leads to persistent viral infection of epitheliums in all systems and organs. SARS only clears the body when the epithelium regenerate.
Guess which epithelium, with the highest concentration of ACE2 receptors also take the longest to regenerate?
Why are some doctors abandoning their rich nations?
Because those rich nations already abandoned their own people and HCW have a bullshit threshold!
Some provincial political leader from a small northern nation I won’t name, last week announced that doctors “needed to step up” to deal with the burden of crisis in children’s hospitals.
The week before they declared financial Wage War against doctors AND nurses!!!
@NjbBari3@farid__jalali All three of you are partially correct. I find it fascinating everyone here is batting around theories of autoimmune disorder when political forces have opened up a population level experiment but nobody is collecting data.
@NjbBari3@farid__jalali Prior to this SARS outbreak the most common infection that lead to lymphopenia was HIV. Now we are seeing varying degrees of autoimmune disease but nobody is applying the simple AIDS tests of t-cell depletion!
@NjbBari3@farid__jalali The trigger for cytokine response and cause of lymphopenia are more likely a result of prion creation of persistent viral infection which damages all systems including immunity.
Candida auris typically infects patients with weakened immune systems. C. auris causes invasive candidiasis (fungemia) in the bloodstream impacting the CNS & multiple organs.
C. auris has attracted attention b/c of its drug resistance, earning the cool name "superbug". But, it's not supposed to infect 600 "weakened immunity" non-patients:
2018-329
2019-466
2020-753
2021-1,765
2022-4,616 (not including this recent outbreak)
A gentle reminder the US/CDC abandoned the WHO's #PrecautionaryPrinciple in 2020. This is the IPC/CDC page for C. auris today, lot's of handwashing, zero isolation of vulnerable patients:
@EvanBlake17 These are interesting development but you seem to be using a oversimplification of ZeroCovid policy and are apparently unaware of medical diagnosis and treatment options recently developed by China.
@EvanBlake17 First, China’s ZeroCovid policy is mostly focused at international travellers and international points of entry. The “Managed Isolation” used by Australia, New Zealand, Hong Kong, Japan & Canada’s Northern Territories are the integral part of ZeroSARS.
@EvanBlake17 I have breaking news; Chinese leadership was draconian before pandemic. But PH measures for pandemic was not draconian. Field hospital for infected is best example, often deemed “draconian”. However, US, UK & EU infected patients in ER next to “not-yet” infected is not draconian?
IFF CH.1.1 continues growth, means it’s a “cousin” mutation where previous antibodies from previous infection or vaccine immunity are being successfully evaded. CH.1.1 will likely outcompete BQ.1.1 (sister) and have concurrent community spread with XBB (or upcoming XBB mutation).
However, the bivalent mRNA vaccine and Novavax vaccine would continue to prevent severe infection, as long as the vaccine activated t-cell are still active. As long as those HCoV t-cells are ACTIVE, there’s active cross-immunity (only prevent death) for all possible SARS variant.