The propaganda is ramping up to pressure admin to open schools. It's based on an interpretation where they look at an exceptional single case in rural wisconsin and ignore the preponderance of evidence about the ability of spread in schools elsewhere.
It's not safe or just to put people in classrooms where they risk long covid, variants, and do not have vaccine protection. If you want kids in classrooms fund the trials and get kids a vaccine. China is doing it. Get with the program! reuters.com/article/us-hea…
putting children in situations where 1/X of them have reactions like this is unfair. We can judge a society by how they treat the infirm, yes, but how about the society which also protects the so called 'least vulnerable?' Covid-19 is not only an acute huffingtonpost.co.uk/entry/long-cov…
illness- these people have a long time to live with whatever unforeseen consequences are in store. So if you want kids in school fast- protect them with a vaccine. If you care about inequality- get them food, mobile internet, and a computer.
A sweeping policy that packs kids- representatives of their families- together in rooms with an aerosol BSL-3 grade pathogen is ridiculous. Ask any virologist if they'd stick their head under a hood with the petri dish despite wearing a mask. How quick we overlook safety
standards to return to the status quo!
More propaganda parade that 'kids are less likely to catch covid-19.' With open schools they are far more likely to catch it.
Funny, they move from absolute risk to relative risk in a different context when it behooves them. Should we remind them of the positivity rates in Texas among schoolchildren? NEGLIGENT. theatlantic.com/ideas/archive/…

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More from @fitterhappierAJ

29 Jan
I've been comparing cov2 to ebola ever since I saw the lymphopenia in Jan in the Lancet. Nasty damn bug. Confuses the hell out of the immune system. theguardian.com/society/2021/j…
I really enjoy looking into Ebola, because when I started out at the NCI we had a phenomenon where we did not know the mechanism
Basically, a talented Physician before I arrived noticed memory T cells in a stimulation would differentiate pools of t cells further pubmed.ncbi.nlm.nih.gov/20971955/
Read 12 tweets
17 Nov 20
I'm going to briefly touch upon one of my favorite subjects: immunological memory. nytimes.com/2020/11/17/hea…
2/ First the B cells: I think for Covid-19 these are the most important to prevent infection and reinfection severity. In this large study on B cell memory, the authors see B cells turning into what we can presume is long-lived memory while continually (for the majorit of people)
3/ shedding Antibodies. Antibody shedding is great because it can control virus that rears its ugly head, and prevent virus from going into a new cell. So that's good. I am worried, though, that there may be cells with a reservoir of competent virus that is influencing this. We
Read 11 tweets
15 Nov 20
@stanleyqilab I think your Crispr package would be well delivered by B cells. They traverse all organs and bone marrow and even play a part in neuroregeneration. Shedding vesicles all the while that can be loaded with RNA. @AnImmunologist @ExcisionBiotx pnas.org/content/117/9/…
Read 4 tweets
24 Sep 20
Back in May I said this paper suggested that orf8 was making it hard for the immune system to clear the virus due to the downregulation of MHC I, and that it forced it to age and compensate for this effect. Compensation can occur with ↑ interferon, the cytokine for inflammation
Recently there was a mutated virus in the wild that showed when the orf8 was nonfunctional, it only caused light illness. I believe that it's light illness bc the immune system no longer has to age and produce interferon so much in order to rec. and clear thelancet.com/journals/lance…
Read 8 tweets
19 Sep 20
After infection you lose A LOT of your naive T cells. Then, not having naive t cells predisposes you to baad outcomes the second go around. I've been saying it. cell.com/cell/fulltext/… Image
Read 5 tweets
9 Sep 20
1/Two things: Given the lack of t cell infiltrates, is this due to a lack of MHC expression on the surrounding cells? This stain should have been done as a mechanistic enquiry, I know canonically neurons do not express it but there is cross presentation by surrounding cells.
2/Perhaps it was and I read it wrong. Unknown; and wondering if the other viruses mentioned also have the leukopenic phenotype this does. The dying people aren't typical cases- there could be reasons they failed to have immune inflitrates, but the finding is significant and
Read 5 tweets

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