Many medicine procedures involve (1) pushing a needle into a fluid-filled space, (2) advancing a plastic catheter over the needle into the space, (3) pulling the needle out, and (4) leaving the catheter in either for drainage or infusion.
Things often go wrong at (2).
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When you first get a “flash” (enter the fluid filled space with your needle), only the very tip/distal part of the needle/bevel is in the target space.
Let’s use paracentesis as an example.
In these photos, the paper is the peritoneum, with views from both sides of it.
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If you try to advance the catheter now, it will hit up against the peritoneum and you will feel resistance. The extra force you exert might even make the needle come out of the peritoneum completely, and you might end up advancing the catheter into subcutaneous tissue.
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What you need to do instead is to advance the *needle* (with the catheter) a little bit more after you get the flash, until the entire bevel and catheter tip are in the desired space. It’s usually just a few more millimeters, like this.
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Now when you hold the needle still and push the white plastic catheter over it, the catheter will go in with minimal resistance, and you can proceed to steps 3 and 4.
Same principle applies to IVs, art lines, central lines, thoras, etc.
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I was taught, and continue to hear it taught, that the history of present illness (HPI) should be crafted to "convince the listener/reader of your suspected diagnosis."
I think this framing is problematic...
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The HPI should outline the patient's experience of illness chronologically, and include with some neutrality details relevant to the *differential* diagnosis.
As well as those symptoms particularly emphasized by the patient (in case you aren't thinking of all differentials).
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Teaching that the HPI should "sell" or "build a case for" a diagnosis essentially encourages us to ignore and hide inconvenient truths that don't fit with our top suspicion, or overemphasize those that do - which is the definition of confirmation bias.
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As you turned the corner on the second flight of stairs, you felt your breath pull a little deeper, the next one come a little earlier. Your heart said 👋🏼, bounding softly in your neck.
Ten seconds down the hall, all that faded. You were back to mulling some thought.
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But hold on. Let’s pause for a minute and retrace the steps.
A lot happened before the extra breath and the tug in your neck caught your attention.
And it’s all so damn cool.
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At the foot of the stairs, anticipation of exertion 🔔 and the stretch of muscle fibers 🦵🏽sent a signal to the sympathetic nervous system: start the car.
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I’ve always thought of severe hypertension as a cause of increased myocardial oxygen demand. Which makes sense for the SBP (afterload, wall stress)... it’s what the LV is contracting against.
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But what role does the DBP play?
Not much of one as far as the LV’s workload far as I can think...
But diastole is when coronary perfusion happens. Applying Ohm’s law in that vascular bed,