1/ YAY Thank goodness for sanity. Statins forum today " Stopped my statins 18 months ago, after having taken them for 28 years. Aside from the constant muscle pains and memory loss, spasms, cataracts, arythmia, etc what finally prompted me to quit is elevated liver enzymes
/2
2/ and the beginnings of kidney problems. My course of action was to cleanse my body from Statins, get my liver in good shape and get a CAC score to see if I do have an actual problem with heart disease despite my elevated Cholesterol.

/3
3/ After all, that's the reason Docs tell us we should be taking Statins in the first place. I got news today that my CAC score is 0.8, heart echo & carotid doppler are perfectly normal. This puts me at a 2.3% risk of a coronary event over 10 years, without Statins!

/4
/4 Thanks to all who have posted in ''Stopped Our Statins'' for giving me courage to stop STATINS and question the status quo of STATIN ' therapy'. Making lifestyle changes, ie low carb, daily execise, stress management, love and appreciate your advice, its a life changer !

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More from @holmanm

7 Oct
1/ Cancer researchers across the world in the last couple of years have found numerous connections between deworming drugs and the decline of cancerous tissue. In 2017, Norwegian scientists found that NTZ, a common anti-parasite drug, killed cancer cells

/2
2/ by decomposing activated beta-catenin. Professor Karl-Henning Kalland of the University of Bergen and leader of the research team who discovered this phenomenon stated, “We discovered that this specific substance is blocking the signaling pathway in the cancer cells…

/3
3/ It is not often that researchers discover a substance that targets specific molecules as precisely as this one,” (“Parasite Killer Too Found to Be Effective Cancer Treatment Candidate,” 2018). Additionally, Kalland’s team discovered that reducing beta-catenin enhanced the

/4
Read 6 tweets
6 Oct
1/ "As always the results of Statin therapy are, to say the least, dramatic.

"In patients with heterozygous FH, moderate- to high-intensity statin therapy lowered the risk for CAD and mortality by 44%".

Wow. But why the need for a composite end point?

/2
2/ If we leave aside soft end points which include coronary re-vascularisation (never influenced by serum lipid levels. No laughing at the back there!) and concentrate on the hard end point of all cause mortality we end up with, for non statinated people:

/3
3/ 9 deaths per 4,892 person-years, which I make 1.8 deaths per 1000 person-years.

On a statin we have 17 deaths per 11,674 person-years, 1.5 per 1000 person-years.

That looks like a reduction in mortality of 0.3 people per 1000 person-years.

/4
Read 7 tweets
26 Sep
1/ Reasons why I don't trust Docs or pharma drugs:
Me age 30, diagnosed with severe Osteoporosis after a bone scan. Nurse told me I had the bones of a 70 yr old. They put me on Bisphosphonates. I researched

/2
2/ these drugs, and realised that they caused necrosis of the jaw, stomach irritation etc. Sooo I decided to do some more research, and what did I find ? Low and behold, Vitamin D3 and Vitamin K2 (MK7) puts calcium where it meant to be, in the BONES not the ARTERIES.
/3
3/ I never looked back. I've had falls from climbing, hard falls, & never broken a bone,. However a friend who has taken Bisphosphonates for the past 2 years recently had an accident. A minor fall from a restaurant stool, which resulted in a very complex break requiring

/4
Read 4 tweets
26 Sep
1/ Here we have Peter Attia discussing the THEORETICAL😂 possibility that lipid-lowering drugs MIGHT negatively impact cognitive function, and specifically, whether PCSK9 inhibitors had such an impact. The answer, according to him, appears to be NO. 🙄

/2
2/ Now I really have to wonder if Attia has some conflict of interest with Amgen, Regeneron and Sanofi since PCSK9’s reduce LDL-C by a massive 50%+. You just have to think - why did he say this ?

/3
/3 Undoubtedly, Statins prevent the body from making an antioxidant that neutralizes the damaging free radicals created by mitochondrial activity. If brain cells—which consume lots of energy—already have mitochondrial problems, then STATIN ‘ therapy’

/4
Read 6 tweets
13 Sep
1/ We show that Statin-induced insulin resistance is a drug CLASS EFFECT. Cerivastatin was removed from the market because of "side effects". Our results show that Cerivastatin was the most potent Statin in promoting adipose insulin resistance.

/2
2/ "We show that statins impaired insulin-stimulated lipogenesis in an adipocyte-autonomous manner, which was prevented by supplementing isoprenoids required for prenylation". WHY ??

/3
3/ because Statins interfere with the Mevalonate pathway & deplete so many organic compounds with great structural diversity and functionality.

- and you can't always put back what Statins stole in the first place. The overriding message is
Stay away from Statins.
Read 4 tweets
9 Sep
1/ Apoptosis (cell death) is an essential component of the maintenance process of our bodies. It is a highly controlled and regulated technique for removing unwanted cells from any organ by the process of induced metabolic suicide.

/2
2/ Tinkering with this process is a potentially catastrophic game. This is the game of Russian Roulette that we play when we take Statins.
That good old law of unintended consequences could have a field day here.

/3
3/ Statins promote apoptosis. They do it in diseased tissues - Cheers for Statins. Unfortunately they ALSO do it in NORMAL tissues too; Boo for Statins.
Read 8 tweets

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