1/ Many of you know me because of my stance on Statin drugs. I have spent many hours over the past 14 years talking to Statin 'victims', trying to find out how they can recover from this drug, and how the drug has destroyed their health - NEEDLESSLY

/2
2/ This lengthy tweet is worth the effort, if it stops just one person going down the Statin route. It is from a dear friend that I got to know while I was on the FB Statins forum. He was 26 years old when prescribed the drug.

/3
3/This is his story: " 19 Years ago yesterday, Oct 11, 2002, I was admitted to UW Madison Hospital in an End-Stage Alzheimer's like state, I could not walk, could not speak coherently, could not feed myself, nor communicate the need to use the restroom, so I wet and soiled

/4
/4 myself for the next 4 weeks. I did not recognize my wife, my 2yr old son, my parents, siblings nor friends who came to visit. I had an MRI scan of my brain, showing multiple scattered lesions throughout the white matter of my brain, I had a brain biopsy revealing holes

/5
5/ in my brain which the doctors considered a spongiform encephalomyopathy, (Human variant of Mad Cow Disease), Electron Microscopy revealed the holes to be Neuronal Apoptosis (Programmed cell death), as well as Mitochondrial DNA mutation most closely

/6
6/ resembling Mitochondrial Encephalomyopathy with Lactic Acidosis and Stroke-Like Episodes (MELAS), the doctors prepared my wife and family for my death until I was evaluated by visiting Professor from Johns Hopkins who started me on a Mitochondrial Cocktail when within

/7
7/ 24 hours of my first dose, I began to be able to recognize my wife, and parents, I could verbalize the need to use the restroom, I was able to answer simple questions, and several days later I was discharged Nov 7 2002, with a diagnosis of "Viral Encephalitis"...I clung

/8
8/ on to that diagnosis because I knew Id be back to work in a few months...or so I thought. A few weeks after discharge, my wife and I were watching Good Morning America, and Diane Sawyer was interviewing Dr. Beatrice Golomb
of the UCSD and was discussing the Statin Effects

9/
9/ Study she was doing, and my wife asked me to enroll, to which I responded "I was a critical care RN, and "knew" everything there was to "know" about these drugs, but thanks to her persistence and power of persuasion, I called the University of California San Diego and

10/
10/ enrolled, had my medical records, including Brain and Muscle Biopsies, MRI scans, etc sent to UCSD. When the study had concluded about 1 year later, I received a phone call from Dr Golomb who informed me that I was one of several patients in her study with strikingly

11/
11/ similar courses of illness, similar biopsy findings etc and she proceeded to tell me she had referred our cases to Dr Doug Wallace PhD of the UC Irvine (whom she referred to as one of the top experts in mitochondrial Disease in the nation, who opined

12/
12/ that my use of LIPITOR was the CAUSAL contributor to the HOLES in my BRAIN, the mitochondrial Mutations, and the peripheral neuropathy. My heart sank as I thought of the hundreds if not thousands of patients I care for I had unknowingly given these drugs (Statins) to.

/13
13/ I fell into a deep depression (which I would never wish on anyone). And that's when I began to spend every spare moment I had into researching the pathophysiology behind this, only to find out we ALL have been suckered into what had undoubtedly become the biggest scam

/14
14/ in the history of the world...the "Lipid Hypothesis".
I told my wife many years ago, that I need to undo the wrongs I did, by doing everything I can to educate everyone as much as I can about the TRUTH, that Cholesterol DOES not, nor ever did cause heart disease.

15/
15/ So thank you everyone for putting up with my constant barrage of Statin/Cholesterol information"

I'd like to add that my friend has never fully recovered from Statin damage. However some "side effects' have been mitigated by stopping the drug sooner rather than later ....

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More from @holmanm

7 Oct
1/ Cancer researchers across the world in the last couple of years have found numerous connections between deworming drugs and the decline of cancerous tissue. In 2017, Norwegian scientists found that NTZ, a common anti-parasite drug, killed cancer cells

/2
2/ by decomposing activated beta-catenin. Professor Karl-Henning Kalland of the University of Bergen and leader of the research team who discovered this phenomenon stated, “We discovered that this specific substance is blocking the signaling pathway in the cancer cells…

/3
3/ It is not often that researchers discover a substance that targets specific molecules as precisely as this one,” (“Parasite Killer Too Found to Be Effective Cancer Treatment Candidate,” 2018). Additionally, Kalland’s team discovered that reducing beta-catenin enhanced the

/4
Read 6 tweets
6 Oct
1/ YAY Thank goodness for sanity. Statins forum today " Stopped my statins 18 months ago, after having taken them for 28 years. Aside from the constant muscle pains and memory loss, spasms, cataracts, arythmia, etc what finally prompted me to quit is elevated liver enzymes
/2
2/ and the beginnings of kidney problems. My course of action was to cleanse my body from Statins, get my liver in good shape and get a CAC score to see if I do have an actual problem with heart disease despite my elevated Cholesterol.

/3
3/ After all, that's the reason Docs tell us we should be taking Statins in the first place. I got news today that my CAC score is 0.8, heart echo & carotid doppler are perfectly normal. This puts me at a 2.3% risk of a coronary event over 10 years, without Statins!

/4
Read 4 tweets
6 Oct
1/ "As always the results of Statin therapy are, to say the least, dramatic.

"In patients with heterozygous FH, moderate- to high-intensity statin therapy lowered the risk for CAD and mortality by 44%".

Wow. But why the need for a composite end point?

/2
2/ If we leave aside soft end points which include coronary re-vascularisation (never influenced by serum lipid levels. No laughing at the back there!) and concentrate on the hard end point of all cause mortality we end up with, for non statinated people:

/3
3/ 9 deaths per 4,892 person-years, which I make 1.8 deaths per 1000 person-years.

On a statin we have 17 deaths per 11,674 person-years, 1.5 per 1000 person-years.

That looks like a reduction in mortality of 0.3 people per 1000 person-years.

/4
Read 7 tweets
26 Sep
1/ Reasons why I don't trust Docs or pharma drugs:
Me age 30, diagnosed with severe Osteoporosis after a bone scan. Nurse told me I had the bones of a 70 yr old. They put me on Bisphosphonates. I researched

/2
2/ these drugs, and realised that they caused necrosis of the jaw, stomach irritation etc. Sooo I decided to do some more research, and what did I find ? Low and behold, Vitamin D3 and Vitamin K2 (MK7) puts calcium where it meant to be, in the BONES not the ARTERIES.
/3
3/ I never looked back. I've had falls from climbing, hard falls, & never broken a bone,. However a friend who has taken Bisphosphonates for the past 2 years recently had an accident. A minor fall from a restaurant stool, which resulted in a very complex break requiring

/4
Read 4 tweets
26 Sep
1/ Here we have Peter Attia discussing the THEORETICAL😂 possibility that lipid-lowering drugs MIGHT negatively impact cognitive function, and specifically, whether PCSK9 inhibitors had such an impact. The answer, according to him, appears to be NO. 🙄

/2
2/ Now I really have to wonder if Attia has some conflict of interest with Amgen, Regeneron and Sanofi since PCSK9’s reduce LDL-C by a massive 50%+. You just have to think - why did he say this ?

/3
/3 Undoubtedly, Statins prevent the body from making an antioxidant that neutralizes the damaging free radicals created by mitochondrial activity. If brain cells—which consume lots of energy—already have mitochondrial problems, then STATIN ‘ therapy’

/4
Read 6 tweets
13 Sep
1/ We show that Statin-induced insulin resistance is a drug CLASS EFFECT. Cerivastatin was removed from the market because of "side effects". Our results show that Cerivastatin was the most potent Statin in promoting adipose insulin resistance.

/2
2/ "We show that statins impaired insulin-stimulated lipogenesis in an adipocyte-autonomous manner, which was prevented by supplementing isoprenoids required for prenylation". WHY ??

/3
3/ because Statins interfere with the Mevalonate pathway & deplete so many organic compounds with great structural diversity and functionality.

- and you can't always put back what Statins stole in the first place. The overriding message is
Stay away from Statins.
Read 4 tweets

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