ICU stories (this story includes the answer to the quiz from yesterday): Young pt w PMH of HTN/HLD/DM2/CAD (stent of obtuse marginal) presented with chest/abd pain, N/V. Stat EKG (infero-lateral "changes"; ST elevation in inferior leads?):
Emergent cath: "diffusely diseased LAD w stenosis 40%, non-dominant Cx with diffuse disease and stenosis <40%, widely patent OM stent, dominant RCA w diffuse disease and stenosis 50%. Pt did not have hemodynamically significant stenosis to explain symptoms and was admitted to CCU
... on nitro drip (for BP control). Next am, pt went into a wide-complex tachycardia that deteriorated in seconds to V fib. CPR started. Defib x1 back to SR. The post-ROSC ECG (that I posted yesterday) showed:
Really ugly looking ☝️, right? An ABG a few min post-ROSC showed normal K (4.1). Pt was intubated and underwent repeat emergent coronary angiogram that showed essentially same findings w the previous night's one. An IABP was placed
POCUS was performed 2 min after ICU arrival. This was the PLAX view (dilated LV; not much contractility in the anteroseptal and inferolateral walls):
PSAX:
4-chamber view (here the base seems to be contracting while the mid- and apical segments not much):
Subcostal:
And a more RV-focused view that was read as "normal" by the - later performed - "formal" echo:
Do you - especially the POCUS-savvy and the cardiologists - agree with the formal echo report that this RV is "normal"?
Next am, during rounds, I took a quick look with POCUS and it was already showing some recovery (subtle?). This was the 4-chamber view:
And the PSAX:
However, pt was oliguric w uop 10 cc/hour. Even though catecholamines are in general contraindicated in this scenario, I decided to try dobutamine drip and see if an ⬆️ in cardiac output would lead to ⬆️ uop. BTW, Vexus was 1. This was the LVOT VTI without and with dobutamine:
LVOT VTI increased from ~ 10 to ~17. There was "contractile reserve". This was the impressive change in contractility shown in POCUS. The 4-chamber view:
And the PSAX:
Despite the increase in CO, the uop did not improve ☹️. After a couple of hours, I stopped the dobutamine drip. If something does not work, why keep doing it? This is Critical Care!
Three days later, LV/RV functions were back to normal. In retrospect, this was a case of Takotsubo cardiomyopathy!
Take-home messages: 1. These funny-looking ECGs for us, the not detail-oriented physicians who have an attention span of 5 sec, should be viewed as potentially secondary to MANY different pathologies. Obviously, we don’t want to miss a case of hyperkalemia, as ...
... it would not be fun to code a hyperkalemic pt in the cath lab (kudos to @EM_RESUS for drawing attention to this scenario). On the other hand, AMI is a time-critical diagnosis (remember “shark fin” in ECG?) and needs to be ruled out ASAP (“time is muscle”).
2. I think Takotsubo cardiomyopathy is a great imitator and can present with a variety of cardiorespiratory or electrocardiographic pictures 3. I don’t know if it’s heresy - since I have not heard it being said that often - but I think it’s fair to say that Takotsubo
cardiomyopathy can co-exist with coronary artery disease 4. POCUS provides a quick + easy way to assess if the therapeutic hemodynamic interventions really accomplish what they are supposed to (for example: do inotropes really increase CO?) and, if not, to be quickly discontinued
5. It's OK to disagree with the formal echo report. The radiologists miss “things” in the x-rays and the CTs. Why do we expect the cardiologists to be flawless? The more experience you get with POCUS, the more you will find yourself questioning the echo reports. That’s fine as
...long as you use these cases as educational opportunities
Thanks for voting (yesterday) and reading today!
BTW, since all good Netflix series end with a final scene full of mystery, this was the patient’s ECG the next day 😊:
ICU stories: Middle-aged pt w PMHx of rheum fever/A fib underwent MV+AV replacement, TV repair w ring, Maze procedure + LA appendage closure. At the end of surgery, TEE was “fine”; pt was transferred to the ICU intubated (fio2 40%) on low-dose levo (0.04). Could not be extubated
because few h later, lactate began to ⬆️ and ivf were given. Levo gtt did not ⬆️ much (just @ 0.1 next am) but lactate was up to 17 mmol/l & pH was 6.98. I was told that pt was probably still "under-resuscitated". When I 👀the chart, pt had received multiple NS, bicarb & albumin
boluses and was > 8 liters positive. I first pulled the bed sheets to look at the legs and feel the skin temp:
The ICU is a place where decisions have to be made frequently and sometimes in a matter of minutes. The phrase "stop iv fluids/start vasopressin/wean norepi to MAP of 70" summarizes multiple orders in a few words. This is a fraction of the orders I placed today in my am shift
Ordering blood count and coags are 2 orders but there are so many other orders that are not documented. It is not far from true to say that an intensivist has to make hundreds of decisions every day in a 15-20 beds' ICU. For those interested, Halpern's group studied intensivist
decision making and how the number/type of decisions are affected by patient, provider, and systems factors. doi: 10.1097/CCM.0000000000001084
If you like making decisions on the fly and titrating pressors while giving orders for a bowel regimen, ICU is your place!
ICU scenarios: it's 5:40 am. After a rapid response is called, the team is bringing to the ICU a 60 yo male pt that has been managed in the COVID-19 ward for 12 ds with NIV/steroids/tocilizumab/empiric antibiotics and anticoag. Pt pulled his mask and desated to the mid-60s...
When he arrived to the ICU, sat was in mid-80s (NIV-Fio2 100%/PEEP10), not much ⬇️ than the last few ds (it was ~90%). He is breathing in the high 20s, in mild/mod resp distress (for whatever that means!). You realize that: i) there is nothing else to offer besides intubation/MV
...and ii) he can probably "go" for a few more hrs without being intubated. You already had a brutal night. You've been up 10.5 hrs. You have no "help" (no resident/fellow/NP/PA). The am crew (MD + PA) arrive at 7 am. Your resp therapists sign-out at 6 am. What would you do next:
Another highlight of my career: Yesterday, the son of an intubated, unvaccinated patient with severe COVID-19 ARDS threatened to sue me because I refuse to administer ivermectin
We usually finish tweets like this with the question “How is your day going?”. But not this time…
To those colleagues that don’t think we’re dealing with a “political”/cult issue: I kindly ask you to think if you had someone in the past threatening you because you did not give a specific drug. This never happened to me + I have been practicing IM -then CCM- for several years
And I never had a sick >vaccinated< patient or his/her family asking specifically for HCQ or ivermectin…
ICU stories: Middle-aged pt with cirrhosis presented to the ED with abd pain and underwent Hartmann's procedure (colectomy - end-colostomy). Next am, pt was hypotensive on rising levo gtt (0.24 from 0.1) and lactate (3.4 -> 6.7). S/he was positive 8 liters in 12 hours
After reading the chart, I was almost certain that pt would be congested/fluid intolerant (after 8 liters+ fluid balance...). When I first walked in the room, BP was 90-100/30-40 (radial a-line), HR 120-130 and this is what the monitor showed:
I did POCUS: hyperdynamic LV, no pericardial effusion, RV OK, IVC very small (images not shown). I threw some color Doppler in the LV and this happened:
ICU stories: Middle-aged pt with cirrhosis presented to the ED with abd pain and underwent Hartmann's procedure (colectomy - end-colostomy). Next am, pt was hypotensive on rising levo gtt (0.24 from 0.1) and ⬆️lactate (3.4 -> 6.7). S/he was positive 8 liters in 12 hours 😱
After reading the chart, I was almost certain that pt would be congested/fluid intolerant (after 8 liters+ fluid balance...). When I first walked in the room, BP was 90-100/30-40 (radial a-line), HR 120-130 and this is what the monitor showed:
I did POCUS: hyperdynamic LV, no pericardial effusion, RV OK, IVC very small (images not shown). I threw some color Doppler in the LV and this happened: