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Dr Naomi Harvey (PhD not MD) #WearAMask Profile picture
@Naomi_D_Harvey
May 19 • 6 tweets • 3 min read Twitter logo Read on Twitter
PI% info @PanickedFoodie @tessfalor

I’m always hot after I eat my evening meal so decided to check my PI and yep, best I’ve ever seen it (usually under 1.5)! Supports eating causing improved blood flow. But…my SPO2 is the lowest I’ve ever seen it. Never seen it go below 98% An oximeter on my finger re...
Only other times I’ve managed to improve my PI% have been my using a hot water bottle on my abdomen (but it only went from 1.5% to 5.7%), or by laying down in bed & heating myself with a hair dryer, where my PI% went over 15% for the first time.
And I’ve been resting different fingers: each finger has different values but my left hand has consistently lower perfusion than my right, which ties in with blood tests where they can never find a good vein on the left!
Repeated it this morning, feeling better today & managed to get dressed (yesterday was my worst day of the year). SPO2 99% again.

PI% 0.6 before breakfast (on right hand with best circulation) raised to 15.8% after breakfast of hot porridge! Oximeter showing a PI% of 0.63Oximeter showing a PI% of 15.8
@Really_Richelle maybe GIP induced?

This improved circulation after eating seems to be helping some people in #TheAcidTest clear high overnight lactate levels, whereas for me, my lactate peaks after breakfast. Got to be two subtypes re: lactate I’d say.
But after a cold lunch of peanut butter on toast & an apple my PI% hasn’t changed at all: 2.4% (which is typical for my right hand). So I haven’t ruled out heat as being the main factor. Oximeter showing a PI of 2.43

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More from @Naomi_D_Harvey

Dr Naomi Harvey (PhD not MD) #WearAMask Profile picture
May 19
Hmmm 🤔 blood tests result pinged in, my phosphate is 0.83mmol/L and the Serum phosphate normal range is 0.83–1.63 mmol/l in healthy adults. Right on the edge of low/not low.
“Phosphate is involved in many critically important biochemical processes including energy metabolism, nucleic acid metabolism, cell signaling, bone formation, and maintenance of acid–base balance”
And this is probably why: “Increased renal clearance of phosphorus occurs in…hyperglycemic states”, which fits with my fasting Glycemia.
Read 5 tweets
Dr Naomi Harvey (PhD not MD) #WearAMask Profile picture
May 18
Hospital trip yesterday about POTS. The doctor was also researcher in blood pressure so he was actually great & was very interested in the data I gave him on my BP, glucose & lactate. Couldn’t offer any help I haven’t already tried but ordered a ton of bloods & a tilt test.
Unfortunately my blood didn’t want to come out & I ended up at the hospital an extra 2 hours & extremely stressed. They finally got some out but as usual the phlebotomist was amazed at how my blood simply wouldn’t come out.
Fully exhausted and felt awful after, hoping the PEM won’t be too bad this coming week now 🤞🏻

As usual despite exhaustion, overdoing it meant I really struggled to sleep 😴 only managed it with a piece of melatonin gummy every few hours when I woke up wide awake.
Read 4 tweets
Dr Naomi Harvey (PhD not MD) #WearAMask Profile picture
Mar 4
Exercise is contraindicated for people with ME/CFS. Full stop.

Those with mild disease may be able to do some gentle exercise, or even occasional long walks, but the body simply doesn’t respond the way it should. Exercise = stress for the body. Healthy bodies adapt, ours don’t.
Cardio exercise especially always carries risk for those with ME/CFS. Even when it’s mild. The usual benefits that healthy people experience from adapting to exercise don’t occur. Cumulative physical stress from regular exercise with ME/CFS may lead to a decline in condition.
Most of us do too much, because we hate being limited, we don’t want to be. But we are.

When I say “exercise is contraindicated” I am referring especially to it being prescribed as treatment.

Movement is important, as much as can be safely done, but movement ≠ exercise.
Read 6 tweets
Dr Naomi Harvey (PhD not MD) #WearAMask Profile picture
Feb 9
Most of us are now familiar with #POTS but there are different types: hypovolemic, neuropathic & hyperadrenergic.

The hyperadrenergic form may be genetically determined & involves blood pressure increase when standing & elevations in adrenaline healthrising.org/blog/2018/08/1…
“Grubb’s 2011 study described hyperadrenergic POTS as having an increase in systolic blood pressure of ≥ 10 mm Hg during a tilt table test with rapid heart beat (tachycardia) or serum norepinephnrine levels that were greater than 600 pg/mL upon standing”
“the key symptomatic differences in hyperadrenergic vs non-hyperadrenergic POTS were increased dizziness, headache, and tremulousness in hyperadrenergic patients.  (Tremulousness refers to trembling, quivering, or shaking hands, voice, etc.)”
Read 8 tweets
Dr Naomi Harvey (PhD not MD) #WearAMask Profile picture
Jan 3
“Compared to uninfected controls, COVID-19 patients of all age groups had severe GSH deficiency, increased OxS and elevated oxidant damage which worsened with advancing age. These defects were also present in younger age groups, where they do not normally occur”
This study looked at 60 people hospitalised with SARS-CoV-2 and found severe glutathione deficiency in them, across all age groups.

Treatment with Glycine plus NAC may help to increase Glutathione and reduce oxidative stress.
I hadn’t looked at Glycine before but it seems highly implicated for post viral illness.

Glycine is essential for central nervous system & connective tissue health.

It also stimulates serotonin production, which may be essential for managing gravity #POTS #MECFS #LongCovid
Read 11 tweets
Dr Naomi Harvey (PhD not MD) #WearAMask Profile picture
Jan 1
In 2003, cardiologist Arnold Peckerman published this paper on #MEcfs using a non-invasive measure of cardiac🫀 output.

It showed patients with severe ME/CFS had significantly lower stroke volume & cardiac output, hence reduced circulation pubmed.ncbi.nlm.nih.gov/12920435/
The level of cardiac impairment correlates with the severity of the disability. Dr Peckerman surmised that those with severe ME/CFS have a form of heart failure (source webmd.com/chronic-fatigu…)
Cardiac output in healthy ppl varies from 7ltrs per min lying down to 5ltrs per min standing. This drop doesn’t affect function. But in ME/CFS the drop may be from 5ltrs lying down to 3.5ltrs standing up. This level of cardiac output causes borderline organ failure.
Read 7 tweets

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