Taking the interest in understanding another's position and where they are coming from is key to having a productive conversation / debate.
But what if someone is persistently misrepresenting your position - even if unintentional?
2/ Of course, the most obvious example of this with clear malice is the Straw Man fallacy (which I won't go into here).
Yet what about where someone restates your position in a way that's not too far off, save some critical flaws that you assume are a simple misunderstanding?
3/ What happens next reveals the problem.
You find you're spending a lot of time attempting to clarify your position in a good faith effort to correct the misunderstanding -- but it isn't working. The other party either isn't getting your original point with repeated attempts...
4/ ... Or it's possible they don't want to.
Regardless, if you're one to (1) start with the assumption each party is acting in good faith and (2) are used to clarifying your position anyway, you're very susceptible to getting caught in this loop. I know I have been...
5/ But there is a simple solution.
... Call out the recasting of your position or statements for exactly that. They may genuinely feel they are summarizing or restating what you're saying in a fair manner - but that's irrelevant to the problem (and you have to recognize this)
6/ So moving forward, I'm going to link this thread when I find I'm that loop.
I'll ask the one I'm chatting with to stick with arguing what I stated as it was stated -- and that I can't spend time constantly correcting their version of it (even if well intentioned).
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First, let me say that data on this has been a bit limited. But *IF* we do ultimately confirm there are more ApoB-48 (B48) than ApoB-100 (B100) in ASCVD plaque, it would be a very big deal.
Let's unpack...
2/ First, thanks to @TuckerGoodrich for pinging me on these pubs and pressing the discussion.
But also credit to @CaloriesProper on tweeting this a couple years ago (I missed it then)
To understand why this would be so important if true, some review...
3/ B48 and B100s are the major proteins on chylomicrons (CMs) and VLDLs, respectively.
CMs mostly carry lipids from the small intestine to the bloodstream (lipids consumed), VLDL mostly carry lipids from storage; predominantly from adipose stores.
#Me: Why would triglyceride rich LDL particles be more atherogenic than triglyceride poor LDL particles?
#ChatGPT: Triglyceride-rich LDL (low-density lipoprotein) particles are more atherogenic (i.e., more likely to contribute to the… twitter.com/i/web/status/1…
2/
#Me: Couldn’t it also be possible that triglyceride rich LDL are ultimately the result of metabolic dysfunction and that better explains its association with atherosclerosis?
#Me: Is it possible that almost the entire amount of atherogenesis associated with high triglyceride rich LDL is due to dysfunctional lipid metabolism and the diseases that result in these profiles rather than the LDL particles themselves?
1/🧵 I'm definitely a fan of both @DominicDAgosti2 and @DrRagnar (obviously), so I was excited to see them chatting about #lipids, #LMHRs, and Dom's consideration of increasing carbs to lower his #ApoB
3/ When chatting with Dom in SD last year for dinner, he mentioned focusing less on maintaining such a sizable muscle mass as he typically does, and I predicted he'd likely see his LDL/ApoB as considerably higher with this change if still #keto. This podcast appears to confirm...
But I also know many people in my own family who have struggled their whole lives to lose weight and assume if they don't have this near instant drop to their goal weight like <fill in the blank success story> then they are doing it all wrong...
... I can't tell you how many times I point out truly new, record-setting successes with people I know personally -- but it's their own record, their own health journey.
But they often can't see it because they hear they "won't be hungry anymore on <fill in blank diet>"...