Suspected COVID-19 because of shortness of breath. Sent to the COVID-19 ED service.
No fever, no cough. No chest pain. Physical exam with patient sitting up (almost 90°):
¿Is this neck pulse arterial or venous? 1/8
Pulse is diffuse and the most striking feature is inward movement. I borrowed this table from @AndreMansoor's must-see lecture on Jugular Venous Pulse **Curiously, notice that there is a single peak instead of the expected double peak 🤔 2/8
I had to get my probe! #POCUS showed severely reduced EF with anterior wall motion abnormality and normal RV function.
EKG showed anterior ST segment changes. This was ACS! Cath lab was activated
3/8
#VExUS showed plethoric IVC, reverse hepatic vein flow and portal vein with >50% pulsatility.
-This was not COVID-19
-Physical exam rules!
-#POCUS definitely enhanced examination
-Single peak on JVP suggests flow reversal (either X or Y descent is missing)
-This may be an interesting cause of diastolic wave reversal on hepatic vein doppler
8/8
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68 yo ♂️ PMH obesity, HTN, CAD w stent, OSA, T2DM
➡️ ED w SOB + fever 39.9°C. Poor oral intake
RR 40, Sat 94% Room Air, BP 157/74 HR 124. Alert. Bibasilar crackles
Labs: Cr 1.3 (baseline 0.8), WBC 10, K 5.4, HCO3 17, CK 184. UA and CXR👇 (case from @NEJM)
How would you manage this AKI initially? What is the likely cause of AKI in patients with #COVID19? (this last question discussed in thread 🧵)
No easy answer except to say that FENa is very unlikely to be useful. It is not unreasonable to try fluids for AKI in the setting of perceived hypovolemia. However, this gets complicated when the potential for worsening ARDS exists. I'll try to tackle the answers one by one 💪
Which of these patients has a more severe degree of venous congestion? #VExUS Thread 🧵 about the Portal Vein (1/17)
Video above shows IVC in short axis, long axis and diameter (from left to right)
Which of these patients has a more severe degree of venous congestion? (2/17)
Abdominal IVC size depends on the difference between CVP and IAP. At a constant IAP, IVC size will increase proportionally to CVP until it reaches the flat part of it's compliance curve. (Great thread by @Thind888 here:
Dr. Gattinoni or: How I Learned to Stop Worrying about P-SILI and Love Furosemide
WEIRD THREAD 🧵 About the blood-gas barrier and #COVID19 (1/9)
Clinical Case: A 4 year old Thoroughbred Horse with a history of recurrent racing-associated epistaxis comes to your office complaining of decreased track performance. He wants to know if there is anyway to prevent this from happening (2/9)
“If you think an awake patient having normal mental status and a basic metabolic panel is available needs a blood gas, you’re wrong. You need the blood gas done on yourself because you may be brain dead.” quote from Dr. Corey Slovis
A few days ago I ran this poll. Most of you chose not to get an ABG. This is in fact what I did (and disappointed the consulting team) Pt had no comorbidities, and HCO3 was normal. (2/7)
Pt not on opioids and no COPD should have a preserved respiratory drive, thus hypercapnia should NOT occur unless exhaustion. Instead, I monitored my pt closely and that same evening O2sat much better. A few days later pt was discharged home. Not a single ABG was drawn. (3/7)