FUNCTIONAL NEUROLOGICAL DISORDER (FND): as the legal profession is want to say, “you can’t prove a negative”. FND is a negative.
Over the course of my career in clinical medicine since 1967, I have encountered Neurologists and other medical specialists on countless occasions. Of course it is not good to generalize, but there seem to be certain characteristics common to Neurologists...
Neurologists are steeped in dogma. I believe that this relates to the primitive tools (by standards of future technology not yet invented) to image and measure neural functions. It is a cliche in clinical medicine that Neurologists can name conditions, but ....
Neurologists have precious understanding of why the nervous system has dysfunctions, to wit the brain is a vast sphere of mysterious activity. One common & favored Neurology tool is the Nerve Conduction Test, NCT, a barbaric instrument that imparts torturing pain to patients.
Nerve Conduction Tests (NCT) are not only painful they are inexact. I was in clinical medicine for a decade or more before a Neurologist informed me a secret I had never heard before. The NCT only becomes positive after a nerve trunk has been dead for two weeks.
Nerve Conduction Tests monitor function of large peripheral nerve trunks. When electrical discharges are released by operator of tests, electromagnetic (EM) fields are so great that function of myriad nerve connections, endings, & neural transducers are hidden behind EM noise.
Neurologist therefore have relatively crude tests of nervous system function. Common symptoms seen in medical practice are dysautonomias: migraine headaches, panic attacks, Gastroparesis, IBS, Interstitial Cystitis, Dysmenorrhea, etc.
Dysautonomias are mediated by autonomic nervous system, which are tissues squarely in sphere of Neurology clinical practice. Unfortunately, analysis of dysautonomias requires in-depth history & hands on analysis of living tissues; efforts not easily fit into 15 min. appts and...
Dysautonomias are not detected by blood tests nor by specialized electronic devices nor imaging studies. Therefore, dysautonomias are not easily discovered by objective.
Dysautonomias reflect intermittent impingements of autonomic nerve & absent nerve cell death; not able to be detected by Nerve Conduction tests (NCT). Often, the osseous & joint subluxations that arouse dysautonomias can be found by in-depth palpation, a lost medical art.
Neurologist, who are often cerebral sorts, often fall back onto the fallacy that, “absence of evidence is evidence of absence”. Psychological theories are convenient facades of explanations for patients. “Functional!”, they explain, while not looking well enough to the tissues.
Dysautonomias aroused by musculoskeletal subluxations are phenomena I have already discussed in several of my threadreaders.
Dysautonomias are effected by imbalances of activation within the two branches of the autonomic nervous system, the Sympathetic and the Parasympathetic. In my next threadreader, I will discuss the use of cannabinoids to induce autonomic neural balance, called “homeostasis”.
Dysautonomias are often reflective of imbalance of activations of the two branches of the autonomic nervous system, the Sympathetic and the Parasympathetic. In my next threadreader, I will discuss use of cannabinoids to correct these imbalances & to induce “homeostasis”.

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More from @BadgleyLaurence

19 Oct
Medical literature reports that every time the female pelvis is surgically invaded (think laparoscopy) adhesions are formed; evoking IBS. Symptoms of Endometriosis align with those of IBS. Endometriosis is simply a name chosen for a symptom constellation in search of a cause.
Even the Gynecology experts admit that cause of Endometriosis is unknown. Could a fomenting insult be widespread estrogen medication/overdose (BCP) of women since 1960’s? Now ~10% of women with Endometriosis! Unnatural!
Could Endometriosis be an iatrogenic disease; a mirage that evolves from Stage I to Stage IV via escalating laparosopic surgeries and repeated tissue ablations; exacerbated by applied unproven hormonal therapies?
Read 4 tweets
13 Oct
UNEQUAL LEGS IN EDS: this is a subject that has titillated my clinical curiosity for many years. I have parked the idea here; so that I can pontificate on the subject over the next several days; as a threadreader chapter for my book.
SHORT LEG & CHRONIC PAIN
In 1972, I was building my first medical practice (San Francisco Bay Area) & heard about a seminar being put on by a famous British Orthopedic Medicine expert, Dr. James Cyriax. I went to his seminar & learned fundamentals of bio-mechanical diagnosis.
Over the years, Physical Therapists & Chiropractors have been reporting a phenomenon called the “Short Leg Sign”, which has been touted as a sign of lower body bio-mechanical disorder & pain generation.
Read 69 tweets
12 Oct
DYSAUTONOMIAS of CARDIOVASCULAR SYSTEM are varied. MCAS & Vagus nerve function are mentioned, but authors chose to overlook common association of Panic Attacks, Mitral Valve Prolapse (MVP), & POTS with Hypermobility Syndrome.
Intriguing physiologic relationships between hypermobile tissues & cardiac dysautonomias? Hypermobility is an inherited tissue variation. Mitral valve & vascular tissues are comprised of connective tissue.
Loss of cardiac mitral valve competence & vascular tone likely potentiate hypotension & syncope. An expected homeostatic response to hypotension is hyperpneic lung efforts to gather more oxygen for blood, i.e., Panic Attacks.
Read 12 tweets
4 Oct
FM & DM
DO FIBROMYALGIA & DIABETES SHARE SAME PATHOPHYSIOLOGY?
@fibromyalgiaME:
“The latest Fibromyalgia - ME - Chronic Pain! paper.li/fibromyalgiaME… Thanks to @item9labs @Travis_Statham @SoIsFibroReal #fibromyalgia #anxiety. Is insulin resistance the cause of Fibromyalgia”
MY RESPONSE: Landmark study or red herring? Study of 23 patients with Fibromyalgia (FM) & small fiber neuropathy(SFN; found in FM & diabetics) showed elevated HgbA1c levels & pain reduction with use of Metformin.
The authors point out the obvious: people with Fibromyalgia are often sedentary due to pain. Sedentary people ➡️ obese. Obesity ➡️ Diabetes, which ➡️ small fiber neuropathy.
Read 21 tweets
3 Oct
ASSOCIATION OF hEDS & RHEUMATOLOGICAL DISORDERS; AN OPINION (1/7/2020):
Either there is genetic linkage between these disparate Syndromes or there is not. I searched for physiologic relationship, but I find no ready theories of associations between these diseases & tissue disorders.
The environmental dynamics that explain these relationships (associations) are sedentary lifestyles that beget weak bodies & chronic pain that forestalls ongoing pursuit of daily locomotor confrontations with the ambient gravitational field.
Read 15 tweets
1 Oct
EDS: I include EDS in the estimated 15% of women I encounter in my medical practice and who have manifest hypermobility. My view is that Mother Nature gave women genes for ligaments rendered more lax, via the hormone Relaxin, to ease the bio-mechanical assaults of childbirth.
The genes for female Hypermobility are sex linked because men are not well served, in their pursuits of war & hunting, by delicate joints of knees, spine, & pelvis.
My encounters with the common expression of Hypermobility amongst Hmong, Mexican, & Hawaiian cultures, recently evolved from a long history of agrarian-subsistence existence, led me to a theory about why hypermobility genes are so highly conserved.
Read 14 tweets

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