VAGINISMUS: is not a psychological problem. When these women are physically examined they are usually found to have laxity of ligaments of one of the sacroiliac joints. Pain generation from this joint subluxation radiates to the mid-pelvis.
There are 16 clinical examinations that physicians can do in the exam room to detect sacroiliac joint subluxation chronic pain disorder syndrome, but most physicians are ignorant of these examinations.
Some of the Signs are: Forward Flexion Test, Fortin Sign, Gillette Sign, Gaeslen’s Sign, Sacral Shear, Pelvic Compression, Thigh Thrust, Active Straight Leg, & the Badgley Book Sign. Another is to have the patient hop up and down on one leg to arouse ipsilateral buttock pain.
When a woman assumes the supine position with thighs flexed and abducted & as the male weight forces the legs apart, the biomechanical forces force the sacroiliac joint to sublux if the joint ligaments are lax.
Unconscious response of female pelvis is widespread muscle spasm in pelvic girdle; muscles contract & splint in attempt to stabilize the joint. Nature abhors loose joints, wherein ligaments are stretched & neutral anatomical lengths are exceeded; very painful events: vaginismus.
Women with hypermobile ligament tissue variations (est. 15%) are prone to develop permanent chronic sacroiliac joint ligament injuries from childbirths, slips and falls, & even from seemingly menial lifting events.
Women with lose sacroiliac joints commonly experience sciatica & restless non-refreshing sleep. No matter what posture pelvic girdle is placed against sleep surface, ground surface forces transmit pelvic ring to loosest joint, stretching it & arousing pain in stretched ligaments.
Another consequence of an unstable pelvis is a thoracolumbar spine given to scoliosis when upright; to maintain a medial center of gravity of the upright body tower. This is common in women with Hypermobility & is a “functional” scoliosis that dissipates when person is reclined.
Problem with a functional scoliosis is that it induces shoulder girdle asymmetry that leads to a slightly tilted head and crossed muscle syndromes (one side hypertrophic from chronic excess weight bearing) in neck, shoulders, and in muscles supporting the spine.
In hypermobile women, crossed muscle syndromes cause widespread chronic muscle spasms & entheses known as the Fibromyalgia trigger points, & these muscle spasms evoke widespread subluxation of body joints (including vertebrae).
Subluxing joints are given to impingement of the autonomic nerve system, which closely approximates the skeletal system. Impinged autonomic nerves evoke a multitude of dysautonomias, which are the hallmarks of Fibromyalgia.
Dysautonomias are known as Migraines, TMJ, TOS, Panic Attacks, Gastroparesis, IBS (both d & c), Interstitial Cystitis, & Dysmenorrhea. I speculate that Endometriosis, IBD (Crohn’s & UC), & CRPS are advanced disorders & consequences of long-term & entrenched dysautonomias.
Fibromyalgia is not one disorder or disease. It is a true Super-Syndrome. One problem with recognition of the causes of Fibromyalgia is that it has multifactorial causes. It manifests variously in different suffers.
Another characteristic that confounds recognition of the etiologies of Fibromyalgia is that the evolution of the tissue disorders & physiological changes occur over months and years; beyond the awareness of most doctors.
Other factors that obscure the etiologic relationships of Fibromyalgia are that the attendant subluxations, muscle spasms, & autonomic neural impingements are unable to be objectively demonstrated with imaging & electrodiagnostic studies.
Surely, there are blood studies (Substance P, cytokines, etc.) & functional MRI studies that are abnormal in Fibromyalgia sufferers. But no scientist can dispute that these abnormalities are not effects, nor can they scientifically assert that these measured functions are causal.
The most scientific way to study & prove etiologies of Fibromyalgia is an in-depth history of life events to discover specific mechanisms of injury accompanied by close view & sensitive palpation of structures of the naked body as the various body parts function in real time.
Static 2-dimensional imaging studies, whether X-ray, CT, or MRI, are relatively valueless compared to the data able to be gathered by the seasoned wise minds of inquisitive clinicians who have had extensive self-training in physical examination of living human tissues.
Unfortunately, the business model of modern medicine and the standard 15 minute appointment has long rung the death knell for true medical diagnosis of disorders of the Super-Syndrome termed Fibromyalgia.
It is a firm precept of the medical arts and sciences that rational therapies and true healing require first the knowledge of the true cause of a disease or disorder. With Fibromyalgia the true causal knowledge has been sorely overlooked.

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More from @BadgleyLaurence

27 Oct
AUTONOMIC CONTROL OF FUNCTIONAL DISORDERS:
There is no need to default to psychological explanations for aberrations of human behavior. Behavior is much controlled by the brain, autonomic nervous system, & hormones, which effect behavior through lens of the brain.
Let’s take bipolar behavior. What tangled psychology might be modeled to explain periodic fluctuating manic/depressive behavior? None has been articulated. Simple explanation is environmental & social activities can stimulate behavioral events via autonomic nervous system (ANS).
Loss of sleep can trigger adaptive behavioral responses via autonomic nerve system & endocrine perturbations. Physical inactivity due to pain can do likewise. Unfortunately, modern clinical sciences lack teachable methods to monitor these perturbations.
Read 50 tweets
23 Oct
FUNCTIONAL NEUROLOGICAL DISORDER (FND): as the legal profession is want to say, “you can’t prove a negative”. FND is a negative.
Over the course of my career in clinical medicine since 1967, I have encountered Neurologists and other medical specialists on countless occasions. Of course it is not good to generalize, but there seem to be certain characteristics common to Neurologists...
Neurologists are steeped in dogma. I believe that this relates to the primitive tools (by standards of future technology not yet invented) to image and measure neural functions. It is a cliche in clinical medicine that Neurologists can name conditions, but ....
Read 15 tweets
19 Oct
Medical literature reports that every time the female pelvis is surgically invaded (think laparoscopy) adhesions are formed; evoking IBS. Symptoms of Endometriosis align with those of IBS. Endometriosis is simply a name chosen for a symptom constellation in search of a cause.
Even the Gynecology experts admit that cause of Endometriosis is unknown. Could a fomenting insult be widespread estrogen medication/overdose (BCP) of women since 1960’s? Now ~10% of women with Endometriosis! Unnatural!
Could Endometriosis be an iatrogenic disease; a mirage that evolves from Stage I to Stage IV via escalating laparosopic surgeries and repeated tissue ablations; exacerbated by applied unproven hormonal therapies?
Read 4 tweets
13 Oct
UNEQUAL LEGS IN EDS: this is a subject that has titillated my clinical curiosity for many years. I have parked the idea here; so that I can pontificate on the subject over the next several days; as a threadreader chapter for my book.
SHORT LEG & CHRONIC PAIN
In 1972, I was building my first medical practice (San Francisco Bay Area) & heard about a seminar being put on by a famous British Orthopedic Medicine expert, Dr. James Cyriax. I went to his seminar & learned fundamentals of bio-mechanical diagnosis.
Over the years, Physical Therapists & Chiropractors have been reporting a phenomenon called the “Short Leg Sign”, which has been touted as a sign of lower body bio-mechanical disorder & pain generation.
Read 69 tweets
12 Oct
DYSAUTONOMIAS of CARDIOVASCULAR SYSTEM are varied. MCAS & Vagus nerve function are mentioned, but authors chose to overlook common association of Panic Attacks, Mitral Valve Prolapse (MVP), & POTS with Hypermobility Syndrome.
Intriguing physiologic relationships between hypermobile tissues & cardiac dysautonomias? Hypermobility is an inherited tissue variation. Mitral valve & vascular tissues are comprised of connective tissue.
Loss of cardiac mitral valve competence & vascular tone likely potentiate hypotension & syncope. An expected homeostatic response to hypotension is hyperpneic lung efforts to gather more oxygen for blood, i.e., Panic Attacks.
Read 12 tweets
4 Oct
FM & DM
DO FIBROMYALGIA & DIABETES SHARE SAME PATHOPHYSIOLOGY?
@fibromyalgiaME:
“The latest Fibromyalgia - ME - Chronic Pain! paper.li/fibromyalgiaME… Thanks to @item9labs @Travis_Statham @SoIsFibroReal #fibromyalgia #anxiety. Is insulin resistance the cause of Fibromyalgia”
MY RESPONSE: Landmark study or red herring? Study of 23 patients with Fibromyalgia (FM) & small fiber neuropathy(SFN; found in FM & diabetics) showed elevated HgbA1c levels & pain reduction with use of Metformin.
The authors point out the obvious: people with Fibromyalgia are often sedentary due to pain. Sedentary people ➡️ obese. Obesity ➡️ Diabetes, which ➡️ small fiber neuropathy.
Read 21 tweets

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