A team member called me to say “We need you at the bedside NOW”, without further details, but with a higher sense of urgency than I’m used to from this person, so I ran to the bedside.
In fact, shortly after arriving my watch asked me if I was starting to exercise
Intentionally obfuscating details about the patient, suffice it to say there was a major arterial bleed into the oropharynx which developed without warning and lead to prompt cardiac arrest.
Fellow quickly prepped to intubate while CPR was ongoing. Due to neck stiffness, DL was not possible, and VL was attempted.
Given the bleeding, we applied liberal suctioning and stayed “high and dry” with the blade so it did not become soiled.
It was frankly impossible to identify any anatomy. While blood could be suctioned to relative clearing of the airway, there were clots etc which obscured identification of even the epiglottis.
A second attempt was planned, at which point I asked for the cric kit
Thanks to prior mental rehearsal (in past situations), I was mentally prepared to cut if needed. I had a moment to gather myself, slow my breathing and get out of “exercise” mode on my watch.
During the 2nd attempt, no improvement in view was made.
The neck was prepped, and I gathered a 10 blade, bougie, and 6.0 ETT alongside the patient’s right (I’m RHD). One more look into airway by diff airway team, no luck.
I performed the procedure near exactly the same as in this video from #AIMEairway
Retrospectively, the ‘hard’ parts (decision to cut, identifying the anatomy, making the right incision) were easier. Bougie glid in easily.
Unanticipated difficulty was passing ETT. I don’t think I made the initial incision into the CTM wide enough. Had to overcome high resist
For reference, see again this video around the 24 sec mark, and how wide she makes the CTM incision after the twist
I’m quite sure I was holding my breath while waiting for the ETCO2 to register confirmation, but it finally did.
In a sad case of “successful procedure, poor outcome”, hemostasis could not be achieved.
My learning points from this case: 1/x
- Again, dilating the CTM sufficiently wide would have made tube passage easier, a lesson for the next time
2/
- It’s difficult to gauge appropriate tube depth when you’re down so low. In the future I might opt for a tracheostomy tube instead to ensure proper depth, and also make it easier to secure
3/
- While it was easier to conclude FONA was needed in this case, I’m glad I didn’t delay
- A few sessions of simulation followed by hundreds (thousands?) of mental rehearsals set me up for success
- Next time this is needed, I won’t hesitate
4/4 - Finally, having the gumption to do this would not have been possible without all the #FOAMed on the topic I’ve read over the past decade. There’s plenty to criticize on this site, but the learning opportunities are invaluable.
Thanks for reading!
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Most of the evidence presented involves critically ill patients, but the lessons shouldn’t much differ if you have a hypotensive/malperfused patient on the wards.
Without the benefit of vasopressors, it’s difficult to determine how to safely resuscitate
We advocate for a hemodynamically-targeted approach to fluid resuscitation. It’s difficult to assess fluid responsiveness in the ICU, moreso on the wards.
🚨 Multi-part case #Tweetorial
24y man no PMH. 3-4 days myalgias, freq diarrhea. 2L saline given on med floor -> hypox/tachypnea. COVID pend.
A 👍
B RR30-34, SpO2 92% 5L NC
❤️ MAP 90, HR 110s ->150s over 12 hrs
D Avpu
E Anxious
L CRP 58 (ULN 0.9), Cr 1.4 (no baseline), Lact 4.8
WOB is moderately elevated.
What diagnostic manuever do you think is most important at this point?
Unlike most MC questions, you also get to choose a therapeutic maneuver while dx is happening. What's your first choice?
First step is to examine the flow waveform. In this case a patient in volume control, to observe how passive expiration is changed by changing time constant.