Matt Siuba Profile picture
28 Jul, 15 tweets, 4 min read
Reflections on a cricothyrotomy:

I wanted to share my personal first experience performing this high-acuity and rare procedure, in the hopes that others will learn from it.

Consider this part reflection, part tweetorial

upload.wikimedia.org/wikipedia/comm… Image
A team member called me to say “We need you at the bedside NOW”, without further details, but with a higher sense of urgency than I’m used to from this person, so I ran to the bedside.

In fact, shortly after arriving my watch asked me if I was starting to exercise
Intentionally obfuscating details about the patient, suffice it to say there was a major arterial bleed into the oropharynx which developed without warning and lead to prompt cardiac arrest.
Fellow quickly prepped to intubate while CPR was ongoing. Due to neck stiffness, DL was not possible, and VL was attempted.

Given the bleeding, we applied liberal suctioning and stayed “high and dry” with the blade so it did not become soiled.

More here: criticalcarenow.com/hyperangulated…
It was frankly impossible to identify any anatomy. While blood could be suctioned to relative clearing of the airway, there were clots etc which obscured identification of even the epiglottis.

A second attempt was planned, at which point I asked for the cric kit
Thanks to prior mental rehearsal (in past situations), I was mentally prepared to cut if needed. I had a moment to gather myself, slow my breathing and get out of “exercise” mode on my watch.

During the 2nd attempt, no improvement in view was made.
The neck was prepped, and I gathered a 10 blade, bougie, and 6.0 ETT alongside the patient’s right (I’m RHD). One more look into airway by diff airway team, no luck.

I performed the procedure near exactly the same as in this video from #AIMEairway

More on this technique from @srrezaie

rebelem.com/bougie-assiste…
Retrospectively, the ‘hard’ parts (decision to cut, identifying the anatomy, making the right incision) were easier. Bougie glid in easily.

Unanticipated difficulty was passing ETT. I don’t think I made the initial incision into the CTM wide enough. Had to overcome high resist
For reference, see again this video around the 24 sec mark, and how wide she makes the CTM incision after the twist

I’m quite sure I was holding my breath while waiting for the ETCO2 to register confirmation, but it finally did.

In a sad case of “successful procedure, poor outcome”, hemostasis could not be achieved.
My learning points from this case: 1/x

- Again, dilating the CTM sufficiently wide would have made tube passage easier, a lesson for the next time
2/
- It’s difficult to gauge appropriate tube depth when you’re down so low. In the future I might opt for a tracheostomy tube instead to ensure proper depth, and also make it easier to secure
3/
- While it was easier to conclude FONA was needed in this case, I’m glad I didn’t delay
- A few sessions of simulation followed by hundreds (thousands?) of mental rehearsals set me up for success
- Next time this is needed, I won’t hesitate
4/4
- Finally, having the gumption to do this would not have been possible without all the #FOAMed on the topic I’ve read over the past decade. There’s plenty to criticize on this site, but the learning opportunities are invaluable.

Thanks for reading!

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More from @msiuba

6 Aug
🚨New #OpenAccess Article 🚨

New review article highlighting key points in fluid resuscitation for septic patients, on the wards or perhaps on the verge of needing ICU.

I admit, not without agenda (⬇️fluid overload)

Freely available through Sept 2: sciencedirect.com/science/articl… ImageImage
A few editorial points:

Most of the evidence presented involves critically ill patients, but the lessons shouldn’t much differ if you have a hypotensive/malperfused patient on the wards.

Without the benefit of vasopressors, it’s difficult to determine how to safely resuscitate
We advocate for a hemodynamically-targeted approach to fluid resuscitation. It’s difficult to assess fluid responsiveness in the ICU, moreso on the wards.

(Proposed approach, not prospectively validated) Image
Read 8 tweets
5 Aug
Phenomenally lucky to have @AndreMansoor speak at our Critical Care Conference today on Diagnostic Reasoning. Image
A few take aways that resonated:

1) A thorough diagnostician doesn’t ask every question/ROS or perform every exam maneuver

2) Order tests/labs in anticipation of a finding (rather than a shotgun “see what comes up” approach)
3) “Everybody knows how to order a cardiac MRI but not how to examine neck veins” 🔥

4) Of course, the frameworks: amazon.com/gp/product/149…
Read 4 tweets
29 Jun 20
🚨 Multi-part case #Tweetorial
24y man no PMH. 3-4 days myalgias, freq diarrhea. 2L saline given on med floor -> hypox/tachypnea. COVID pend.
A 👍
B RR30-34, SpO2 92% 5L NC
❤️ MAP 90, HR 110s ->150s over 12 hrs
D Avpu
E Anxious
L CRP 58 (ULN 0.9), Cr 1.4 (no baseline), Lact 4.8 Image
WOB is moderately elevated.

What diagnostic manuever do you think is most important at this point?
Unlike most MC questions, you also get to choose a therapeutic maneuver while dx is happening. What's your first choice?

More case details to follow tomorrow!
Read 25 tweets
13 May 20
🚨Crashing Patient Case🚨

72 yo ♀️ to ED with syncope, hypotension. Intubated for hypercap/hypox resp failure. Hypotensive post, tx to ICU.

A Tubed
B FiO2 1.0, 10 ml/kg Vt
❤️ MAP 70 -> 50 -> 30 in mins
D sedate, +cough
E Leaky 22g PIV
U n/a
L WBC 35k, lact 1.7, UA+CXR shown
First step?
My team is very adept at CVCs, but we go for the IO (1 min to insert vs 10, and much higher first past success doi.org/10.1097/ccm.00…)

More on IOs from LITFL litfl.com/intraosseous-a…

Also, the vent is alarming for high peak pressures (45 cm H2O consistently). What next?
Read 17 tweets
21 Apr 20
A case in the age of #COVID19

A 50 yo man with no PMH came to ED with confusion & hypoxemia. Admission CXR shown. Possible COVID+ contact.

A Protecting
B RR 28 SpO2 92% 60% HFNC
C BP 90/65 HR 105, Cap Ref 4 s
D A&0 x 4
E - T 37.9
L CRP 3x ULN, WBC 21 (PMNs)

#Tweetorial Image
On arrival to the ICU he develops shock requiring vasopressors. Abx staretd. Mottling on knees. What's the next step?

Case details to follow tomorrow!
ABG in ED 7.38 / 34 / 70 / 21 on 5L NC in ED, I did not repeat on HFNC.

IL-6 level 120 (normal: <6)

https://t.co/iDvQjOUp6R
Read 13 tweets
9 Apr 20
Interesting issue in mechanical ventilation of #COVID19 patients, each one here after >10 days of mechanical ventilation.

How might we monitor for development of this? Time to pay attention to resistance, oft neglected in the age of COVID #ARDS...

#tweetorial #SoMe4MV
First step is to examine the flow waveform. In this case a patient in volume control, to observe how passive expiration is changed by changing time constant.

More on time constants here: derangedphysiology.com/main/cicm-prim…
A faster time constant is associated with decreased compliance (increased elastic recoil). The opposite is seen with airway resistance.

In these cases we often see the expiratory phase on the flow diagram *not* return to baseline before the next inspiration is initiated
Read 9 tweets

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