CT shows opacification of the frontal and anterior ethmoid sinuses without evidence of cortical dehiscence
💡 In peds, infection can spread through vascular channels w/o destroying the bone
MR shows abnormal signal filling the sinuses with associated restricted diffusion 2/2 purulent material
Post contrast we see areas of relative hypoenhancement in comparison to the normal mucosa on the contralateral side
There is smooth pachymeningeal enhancement, suggestive of intracranial extension as well as an abscess in the right frontal lobe
Learning points:
💡 Acute sinusitis is a clinical diagnosis (imaging has poor correlation w/ symptoms as there is a high incidence of mucosal abnormalities in asymptomatic patients)
💡 Even normal sinus CT can have endoscopic evidence of sinusitis
💡 imaging can be used when complications are suspected
💡 CT sinuses can also be used in the outpatient setting in patients with chronic or recurrent sinusitis as a preoperative evaluation in those who may benefit from FESS
💡 Reduced or absent mucosal enhancement is NOT pathognomonic for invasive fungal sinusitis as it can be seen in mucoceles (possibly with superimposed infection) and patchy asymmetric diminished enhancement in bacterial sinusitis
Possible mechanism is increased sinus pressure leading to reduced perfusion or possible mucosal necrosis (if it can invade bone why not cause mucosal necrosis)? This is just speculation, I do not know what actually occurs 🤷🏻♂️
💡 Invasive fungal is usually thickened absent mucosal enhancement, no restricted diffusion, bone erosions and immunocompromised host
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Brain radiation is a risk factor for the development of meningiomas …this patient subsequently developed multiple meningiomas including this large frontal meningioma
The mass was treated and immediate post op CT shows expected post op change with the resection cavity, some hemorrhage, and pneumocephalus
▶️Initial head CT shows subarachnoid hemorrhage centered in the right cerebellopontine angle cistern
▶️CTA confirms an aneurysm of the right anterior inferior cerebellar artery (AICA)
▶️MR displays and ice cream shaped enhancing mass extending through the right internal auditory canal into the cerebellopontine angle cistern, consistent with a vestibular schwannoma #icecream
▶️Careful search into the history confirms the schwannoma was treated with radiation
Answer: confirmed germinoma, all these masses are on the differential for a pineal region mass …perhaps the most helpful clue is the age and gender rather than the imaging 🧠
Some correlates for high grade astrocytoma include necrosis, marked mass effect, restricted diffusion (often patchy, eccentric or nodular in glioblastoma), elevated CBV, enhancement, and older age
The presence of ependymal spread is another poor prognostic sign (shown below)
Important learning point: when dealing with glioblastoma, the neoplasm extent is not defined by the area of enhancement but rather by the entire area of T2/FLAIR abnormality. Microscopic infiltrating tumor clearly extends beyond the areas of enhancement. 🧠
Is it sellar based? suprasellar (S)? clival? Planum sphenoidale (PS)? Tuberculum sellae (arrow)?
▶️Location can change operative approach including but not limited to subfrontal vs transsphenoidal
2️⃣Where is the normal pituitary gland?
▶️this can be tough when the mass is large but the normal tissue often enhances more avidly than tumor so look for a strip of relatively avid enhancement along the periphery of the mass. Surgeons do not want to remove normal pit tissue