DDX:
Must differentiate from infection (as calcific tendinitis is treated with time and NSAIDs)
▶️For infection, expect a more spherical or convex retropharyngeal collection, more enhancement, and presence of head and neck infection
▶️Calcifications at the C1-C2 junction are pathognomonic for calcific longus colli tendonitis 🧠
Companion cases:
▶️Radiograph showing prevertebral swelling and amorphous calcs on C1-C2 junction
▶️Appearance on CT in a different patient showing the amorphous calcs and edema
Companion case 3 (DJD fake out!!):
▶️CT shows COARSE calcifications at C1-C2 junction with mild pharyngeal edema (from prior radiation in this case)
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▶️Susac syndrome is a microangiopathy (likely autoimmune affecting the precapillary arterioles) with a strong female predilection, typically occurring in women age 20-40
Clinical presentation:
Classic triad
1️⃣Encephalopathy
2️⃣Branch retinal artery occlusions
3️⃣Hearing loss
💡Though most patients do not present with the complete triad (it may develop over years)
▶️Biopsy showed a reactive and reparative osseous process and bone culture grew oral flora (though cultures are usually negative)
▶️SOG is thought to be due to a low grade infection possibly 2/2 dental disease. However, there should be no signs of acute infection (suppuration, bony sequestration or draining tracts)
▶️Initial non-con CT shows a 3cm hyperdense lobulated extra-axial mass in the expected region of the left MCA bifurcation, consistent with a giant aneurysm. There are associated peripheral calcifications
▶️ What is the cause of the surrounding hypodensity?