Recent well liked threads
To understand the ET situation on Earth, as well as many other disclosure topics, you must start with the basics.
Why are we here?
We're Souls having a human experience by choice.
Human bodies were created for the purpose of soul incarnation (body by Elohim, Soul by God-Source)
Why are we here?
We're Souls having a human experience by choice.
Human bodies were created for the purpose of soul incarnation (body by Elohim, Soul by God-Source)
What is the reason for suffering and struggle?
Why do athletes use weights? Resistance builds strength.
We are in a duality matrix of free will choices. It's a school for soul learning, not a prison. Individual free will tests were planned for the greatest benefit of humanity.
Why do athletes use weights? Resistance builds strength.
We are in a duality matrix of free will choices. It's a school for soul learning, not a prison. Individual free will tests were planned for the greatest benefit of humanity.
There are positive ETs who have been with us since the beginning. Some look human and some don't. Some have been called "Angels". They serve as our spirit guides and incarnate as humans too, choosing to forget.
Many of us are their Soul Family and come from the same Soul Origin.
Many of us are their Soul Family and come from the same Soul Origin.
In 2021, Steve Bannon distributed a trailer for a purported documentary about Jeffrey Epstein.
Bannon got 15 hours of footage of the pedophile, including interviews in his Manhattan mansion.
What happened to the "documentary"?
In reality, sources told me, it never existed.
🧵
Bannon got 15 hours of footage of the pedophile, including interviews in his Manhattan mansion.
What happened to the "documentary"?
In reality, sources told me, it never existed.
🧵
Bannon's explanation that he was producing a documentary about Epstein was nonsense, according to people who spent time with both men around the time they were in each other's lives.
In reality, the two were friends.
businessinsider.com/steve-bannon-f…
In reality, the two were friends.
businessinsider.com/steve-bannon-f…
I've been trying to see the purported "documentary" for years. And Bannon talks to journalists all the time.
But when I contacted him and his spokesperson and said I was working on a story about his Epstein tapes, I got silence.
businessinsider.com/steve-bannon-f…
But when I contacted him and his spokesperson and said I was working on a story about his Epstein tapes, I got silence.
businessinsider.com/steve-bannon-f…
Most People Have NO IDEA Why They’re Even Alive‼️
Your job isn’t your Purpose.
Your comfort isn’t your Calling.
You weren’t made to Blend In.
So, What Is Your Purpose - as a Christian?
What does it really mean to Walk With God EVERY DAY?
This will change how you live 👇
Your job isn’t your Purpose.
Your comfort isn’t your Calling.
You weren’t made to Blend In.
So, What Is Your Purpose - as a Christian?
What does it really mean to Walk With God EVERY DAY?
This will change how you live 👇
1️⃣
You weren’t created Just To Exist.
You were made TO WALK WITH GOD—Daily, Deeply, Completely.
🙏 “Fear God and keep His commandments,
for this is man’s all.”
—Ecclesiastes 12:13
That’s Your Purpose. Period‼️
You weren’t created Just To Exist.
You were made TO WALK WITH GOD—Daily, Deeply, Completely.
🙏 “Fear God and keep His commandments,
for this is man’s all.”
—Ecclesiastes 12:13
That’s Your Purpose. Period‼️
2️⃣
Walking with God is NOT just about going to a church building and checking a box.
It’s about living IN CONSTANT AGREEMENT with Him.
Talking with Him.
Trusting Him.
Obeying Him.
🙏 “Can two walk together, unless they are agreed?”
—Amos 3:3
Walking with God is NOT just about going to a church building and checking a box.
It’s about living IN CONSTANT AGREEMENT with Him.
Talking with Him.
Trusting Him.
Obeying Him.
🙏 “Can two walk together, unless they are agreed?”
—Amos 3:3
Most people missed it.
A study just showed something absolutely wild:
A single CRISPR infusion cut people’s cholesterol and triglycerides in half.
Yes - half.
From one treatment.
And if this holds up?
Heart disease may never look the same again.
Let me explain:
The target was a gene called ANGPTL3 — a tiny switch in your liver that controls how much LDL (“bad” cholesterol) and triglycerides float in your blood.
Some people are born with this gene naturally turned off.
They live their whole lives with ultra-low lipids.
And they almost never get heart disease.
Nature gave us the blueprint.
So researchers did something bold:
They used CRISPR–Cas9 to recreate that natural mutation.
Not a pill.
Not an injection every 2 weeks.
Not a lifetime of medication.
Just one IV infusion designed to permanently switch off ANGPTL3.
The results?
In the highest-dose group:
LDL dropped ~50%
Triglycerides dropped ~55%
After just 60 days
From a single treatment.
Let that sink in.
This isn’t controlling cholesterol.
It’s rewiring the biology that creates it.
And yes - this all happened in humans.
Not mice.
Not petri dishes.
Not hype.
15 real patients with severe lipid disorders got the therapy.
Is it early? Extremely.
But it’s the strongest signal so far that gene editing can target a common disease - not just rare ones.
Why does this matter?
Because cardiovascular disease is still the world’s #1 killer.
We already have statins, PCSK9 inhibitors, lifestyle programs…
But millions still can’t reach safe levels, can’t tolerate drugs, or can’t sustain lifelong treatment.
A “one-and-done” therapy changes the entire game.
Even the clinicians were stunned.
“We can move from chronic therapy to something that’s one-and-done.” - Luke Laffin, Cleveland Clinic
Imagine telling a patient:
“You’ll never need cholesterol medication again.”
That’s the scale of what’s being tested here.
But here’s the catch - and it’s a big one:
This is very early science.
15 people treated.
Only ~2 months of data.
Long-term risks unknown.
CRISPR editing inside the body.
Off-target effects must be monitored.
Large trials will require thousands of participants
No honest scientist will promise miracles yet.
The real shift is this:
For the first time, gene editing isn’t just for rare disorders.
It’s stepping into the diseases that kill millions.
Ten years ago, investors told researchers this would never happen.
Too risky. Too expensive. Too ambitious.
Now?
Dozens of companies are racing to bring CRISPR to heart disease. The field flipped.
The future is suddenly visible:
CRISPR for high blood pressure.
CRISPR for metabolic diseases.
CRISPR for common cancers.
CRISPR for high cholesterol.
CRISPR for heart failure.
CRISPR for diabetes.
We’re watching the beginning of gene editing as mainstream medicine.
Not science fiction.
Science translation.
If the safety data holds…
If the lipid reductions are durable…
If the therapy becomes affordable…
Then the world’s #1 killer - heart disease - might finally meet a challenger stronger than any pill.
And it started with 15 volunteers and one gene.
That’s the story:
CRISPR isn’t just fixing rare diseases anymore.
It’s coming for the diseases almost everyone has.
The next decade of medicine is going to look nothing like the last.
A study just showed something absolutely wild:
A single CRISPR infusion cut people’s cholesterol and triglycerides in half.
Yes - half.
From one treatment.
And if this holds up?
Heart disease may never look the same again.
Let me explain:
The target was a gene called ANGPTL3 — a tiny switch in your liver that controls how much LDL (“bad” cholesterol) and triglycerides float in your blood.
Some people are born with this gene naturally turned off.
They live their whole lives with ultra-low lipids.
And they almost never get heart disease.
Nature gave us the blueprint.
So researchers did something bold:
They used CRISPR–Cas9 to recreate that natural mutation.
Not a pill.
Not an injection every 2 weeks.
Not a lifetime of medication.
Just one IV infusion designed to permanently switch off ANGPTL3.
The results?
In the highest-dose group:
LDL dropped ~50%
Triglycerides dropped ~55%
After just 60 days
From a single treatment.
Let that sink in.
This isn’t controlling cholesterol.
It’s rewiring the biology that creates it.
And yes - this all happened in humans.
Not mice.
Not petri dishes.
Not hype.
15 real patients with severe lipid disorders got the therapy.
Is it early? Extremely.
But it’s the strongest signal so far that gene editing can target a common disease - not just rare ones.
Why does this matter?
Because cardiovascular disease is still the world’s #1 killer.
We already have statins, PCSK9 inhibitors, lifestyle programs…
But millions still can’t reach safe levels, can’t tolerate drugs, or can’t sustain lifelong treatment.
A “one-and-done” therapy changes the entire game.
Even the clinicians were stunned.
“We can move from chronic therapy to something that’s one-and-done.” - Luke Laffin, Cleveland Clinic
Imagine telling a patient:
“You’ll never need cholesterol medication again.”
That’s the scale of what’s being tested here.
But here’s the catch - and it’s a big one:
This is very early science.
15 people treated.
Only ~2 months of data.
Long-term risks unknown.
CRISPR editing inside the body.
Off-target effects must be monitored.
Large trials will require thousands of participants
No honest scientist will promise miracles yet.
The real shift is this:
For the first time, gene editing isn’t just for rare disorders.
It’s stepping into the diseases that kill millions.
Ten years ago, investors told researchers this would never happen.
Too risky. Too expensive. Too ambitious.
Now?
Dozens of companies are racing to bring CRISPR to heart disease. The field flipped.
The future is suddenly visible:
CRISPR for high blood pressure.
CRISPR for metabolic diseases.
CRISPR for common cancers.
CRISPR for high cholesterol.
CRISPR for heart failure.
CRISPR for diabetes.
We’re watching the beginning of gene editing as mainstream medicine.
Not science fiction.
Science translation.
If the safety data holds…
If the lipid reductions are durable…
If the therapy becomes affordable…
Then the world’s #1 killer - heart disease - might finally meet a challenger stronger than any pill.
And it started with 15 volunteers and one gene.
That’s the story:
CRISPR isn’t just fixing rare diseases anymore.
It’s coming for the diseases almost everyone has.
The next decade of medicine is going to look nothing like the last.
When Shohei Ohtani was a high school freshman, he created a detailed "dream sheet" with one central goal: to be the #1 draft pick for 8 NPB (Nippon Professional Baseball) teams.
It was a 64-cell roadmap based on a framework called the Harada Method.
Here's exactly what Shohei did 👇
1. First, some history.... The Harada Method was created by Takashi Harada, a Japanese junior high track coach. He took a team ranked last out of 380 schools and, using his system, turned them into the #1 team in the region within 3 years. They held that top spot for the next 6 years.
2. You start by placing your main goal in the center of an 8x8 grid. For Ohtani, this was "be the #1 draft pick."
3. Next, you identify 8 critical supporting pillars needed to achieve that goal. These surround the main goal.
Ohtani's 8 pillars were:
• Body
• Control
• Sharpness
• Speed
• Pitch Variance
• Personality
• Karma/Luck
• Mental Toughness
4. You then break down each of those 8 pillars into 8 smaller, actionable tasks or daily routines.
This fills out the entire 64-cell grid, turning a massive dream into a concrete, daily action plan.
To improve his karma, he listed tangible actions like:
• Showing Respect to Umpires
• Picking up trash
• Being positive
• Being someone people want to support
5. The method goes far deeper than just technical skills. It forces you to analyze your weaknesses and build confidence. It also has a highlight on service to others, emphasizing that humility and contributing to your community are essential for personal success.
6. The key to the system is daily execution and accountability. Once the 64-cell chart is complete, you turn the tasks and habits into a daily diary and a "Routine Check Sheet." It’s designed to transform abstract intentions into a measurable, daily practice.
It was a 64-cell roadmap based on a framework called the Harada Method.
Here's exactly what Shohei did 👇
1. First, some history.... The Harada Method was created by Takashi Harada, a Japanese junior high track coach. He took a team ranked last out of 380 schools and, using his system, turned them into the #1 team in the region within 3 years. They held that top spot for the next 6 years.
2. You start by placing your main goal in the center of an 8x8 grid. For Ohtani, this was "be the #1 draft pick."
3. Next, you identify 8 critical supporting pillars needed to achieve that goal. These surround the main goal.
Ohtani's 8 pillars were:
• Body
• Control
• Sharpness
• Speed
• Pitch Variance
• Personality
• Karma/Luck
• Mental Toughness
4. You then break down each of those 8 pillars into 8 smaller, actionable tasks or daily routines.
This fills out the entire 64-cell grid, turning a massive dream into a concrete, daily action plan.
To improve his karma, he listed tangible actions like:
• Showing Respect to Umpires
• Picking up trash
• Being positive
• Being someone people want to support
5. The method goes far deeper than just technical skills. It forces you to analyze your weaknesses and build confidence. It also has a highlight on service to others, emphasizing that humility and contributing to your community are essential for personal success.
6. The key to the system is daily execution and accountability. Once the 64-cell chart is complete, you turn the tasks and habits into a daily diary and a "Routine Check Sheet." It’s designed to transform abstract intentions into a measurable, daily practice.
By the way, this is the exact process we're teaching at Texas Sports Academy for our elite middle school athletes.
Our coaches have the kids...
1. Write down their dream
2. Work backwards to define the necessary habits
3. Build sustainable systems that make execution automatic
We're training our kids to obsess over the process, not the outcome.
We're launching hundreds of schools across Texas next year.
Our coaches have the kids...
1. Write down their dream
2. Work backwards to define the necessary habits
3. Build sustainable systems that make execution automatic
We're training our kids to obsess over the process, not the outcome.
We're launching hundreds of schools across Texas next year.
Just got back from Mauritius. 5 days. Self-planned. No rushing between tourist spots. Just picked one place and enjoyed the most. If you wondering about Flight, Visa, Currency, Car Rental and all other details, here's the complete breakdown 🧵
FLIGHTS: Chennai → Mauritius via Bangalore ₹75k for 2 people (round trip)
Direct flights:
Delhi/Mumbai: Daily
Bangalore: Tue, Thu, Sat, Sun
Chennai: Only Tuesday
6 hours from Bangalore to Mauritius.
Direct flights:
Delhi/Mumbai: Daily
Bangalore: Tue, Thu, Sat, Sun
Chennai: Only Tuesday
6 hours from Bangalore to Mauritius.
VISA: Not needed for Indians. You just need to fill online travel declaration:
Keep Return tickets and Hotel booking confirmation ready for Immigration checks. That's it.safemauritius.govmu.org
Keep Return tickets and Hotel booking confirmation ready for Immigration checks. That's it.safemauritius.govmu.org
大部分产品死于没用户，不是产品不够好。
花 6 个月打磨产品，上线后发现就几个注册，不是朋友就是亲人、还有一些误点进来的。这可能不是你的问题，是方法错了。
看完《拉新》这本书，我尝试整理了一套更有效的实战手册。适合独立开发者和自媒体，都是具体打法，没废话！
先说个反直觉的事实：
花 6 个月打磨产品，上线后发现就几个注册，不是朋友就是亲人、还有一些误点进来的。这可能不是你的问题，是方法错了。
看完《拉新》这本书，我尝试整理了一套更有效的实战手册。适合独立开发者和自媒体，都是具体打法，没废话！
先说个反直觉的事实：
1）50%法则（最重要）
50%时间做产品，50%时间找用户。
不是先做好产品再推广，是边做边测试渠道。
为什么？
因为可能你正在「往破桶里倒水」。产品有漏洞，早期用户会告诉你哪里漏了，你想要 修补→测试→再修补。
Mint 在产品发布前就积累了几万用户，6 个月后破百万。
怎么做的？一边开发，一边测试博客软文、SEO、社交广告。
如果你现在 100% 时间在写代码/做内容？停下来！
50%时间做产品，50%时间找用户。
不是先做好产品再推广，是边做边测试渠道。
为什么？
因为可能你正在「往破桶里倒水」。产品有漏洞，早期用户会告诉你哪里漏了，你想要 修补→测试→再修补。
Mint 在产品发布前就积累了几万用户，6 个月后破百万。
怎么做的？一边开发，一边测试博客软文、SEO、社交广告。
如果你现在 100% 时间在写代码/做内容？停下来！
2）靶心框架（找到你的核心渠道）
别无脑乱试，可以用这个三步框架：
外环 - 列出所有可能
在表格里列出 19 种渠道（下面会讲），针对每个写下：如果成功会是什么样子？需要什么资源？
别跳过任何一个。你觉得不可能的，或许往往最有效。
中间环 - 小成本测试 3-5 个
选最有希望的 3-5 个渠道，快速测试。
标准：1000 美元以内，1 个月以内，回答三个问题：
- 获取一个用户多少钱？
- 能获取多少用户？
- 这些用户是我要的吗？
内环 - 全力搞定一个
找到最有效的那个，然后 all in ！
不要同时做三个「还不错」的渠道，专注一个「很好」的，其他渠道只用来辅助核心渠道。
DuckDuckGo 试了软文、SEO、社交广告、电视广告。但每个阶段只专注一个核心渠道，用完再换下一个。
别无脑乱试，可以用这个三步框架：
外环 - 列出所有可能
在表格里列出 19 种渠道（下面会讲），针对每个写下：如果成功会是什么样子？需要什么资源？
别跳过任何一个。你觉得不可能的，或许往往最有效。
中间环 - 小成本测试 3-5 个
选最有希望的 3-5 个渠道，快速测试。
标准：1000 美元以内，1 个月以内，回答三个问题：
- 获取一个用户多少钱？
- 能获取多少用户？
- 这些用户是我要的吗？
内环 - 全力搞定一个
找到最有效的那个，然后 all in ！
不要同时做三个「还不错」的渠道，专注一个「很好」的，其他渠道只用来辅助核心渠道。
DuckDuckGo 试了软文、SEO、社交广告、电视广告。但每个阶段只专注一个核心渠道，用完再换下一个。
🚨🚨🚨The Unelected LUNATIC, CORRUPT Tyrant von der LIAR Will Provoke WW3
🚨Putin Will Fail – And Europe Will Stand: Von der Leyen: This Winter Will Shape the Future of the War
🚨Putin Will Fail – And Europe Will Stand: Von der Leyen: This Winter Will Shape the Future of the War
Are you guys still using Bloomberg?
You can now use Perplexity AI to track markets, break down earnings, and forecast trends all with one prompt.
Let me giveaway my mega prompt to help you become a pro analyst ↓
You can now use Perplexity AI to track markets, break down earnings, and forecast trends all with one prompt.
Let me giveaway my mega prompt to help you become a pro analyst ↓
Here's the prompt:
"You are my AI financial research analyst.
Your job:
Act as a Bloomberg terminal + McKinsey consultant hybrid.
I’ll give you a company, sector, or theme — you’ll produce institutional-grade research reports.
Your output format must always include:
1. EXECUTIVE SUMMARY
- Core insights in bullet points (5-8 max)
- Key metrics and recent trends
2. COMPANY OVERVIEW
- Core business model, revenue streams, valuation
- Latest financials, growth rates, P/E, debt ratios
3. MARKET CONTEXT
- Competitive landscape and positioning
- Key macroeconomic or regulatory drivers
- Industry tailwinds/headwinds
4. RECENT DEVELOPMENTS
- M&A activity, funding, leadership changes, partnerships
- Recent filings (10-Q, 10-K, S-1) insights
5. SENTIMENT & NEWS FLOW
- Analyst upgrades/downgrades
- Media sentiment (positive/negative/neutral)
- Major events impacting stock price
6. AI SYNTHESIS
- 5 key takeaways investors should know
- 3 action ideas (buy/hold/sell rationale)
- 2 contrarian insights missed by mainstream coverage
Formatting:
- Use concise paragraphs and data-backed statements.
- Include links to credible financial sources (e.g., SEC filings, Reuters, company reports).
- Prioritize insight density over filler.
- When I ask for comparisons, use a side-by-side table format.
Tone:
Objective, precise, and analytical — like a Goldman Sachs or Morgan Stanley equity analyst.
Example query:
“Analyze NVIDIA vs AMD Q3 2025 performance and AI hardware dominance.”"
"You are my AI financial research analyst.
Your job:
Act as a Bloomberg terminal + McKinsey consultant hybrid.
I’ll give you a company, sector, or theme — you’ll produce institutional-grade research reports.
Your output format must always include:
1. EXECUTIVE SUMMARY
- Core insights in bullet points (5-8 max)
- Key metrics and recent trends
2. COMPANY OVERVIEW
- Core business model, revenue streams, valuation
- Latest financials, growth rates, P/E, debt ratios
3. MARKET CONTEXT
- Competitive landscape and positioning
- Key macroeconomic or regulatory drivers
- Industry tailwinds/headwinds
4. RECENT DEVELOPMENTS
- M&A activity, funding, leadership changes, partnerships
- Recent filings (10-Q, 10-K, S-1) insights
5. SENTIMENT & NEWS FLOW
- Analyst upgrades/downgrades
- Media sentiment (positive/negative/neutral)
- Major events impacting stock price
6. AI SYNTHESIS
- 5 key takeaways investors should know
- 3 action ideas (buy/hold/sell rationale)
- 2 contrarian insights missed by mainstream coverage
Formatting:
- Use concise paragraphs and data-backed statements.
- Include links to credible financial sources (e.g., SEC filings, Reuters, company reports).
- Prioritize insight density over filler.
- When I ask for comparisons, use a side-by-side table format.
Tone:
Objective, precise, and analytical — like a Goldman Sachs or Morgan Stanley equity analyst.
Example query:
“Analyze NVIDIA vs AMD Q3 2025 performance and AI hardware dominance.”"
Results:
I’m not sharing this as the CEO of Eternal, but as a fellow human, curious enough to follow a strange thread. A thread I can’t keep with myself any longer.
It’s open-source, backed by science, and shared with you as part of our common quest for scientific progress on human longevity.
Newton gave us a word for it. Einstein said it bends spacetime. I am saying gravity shortens lifespan.
Read on, and tell me what you think.
It’s open-source, backed by science, and shared with you as part of our common quest for scientific progress on human longevity.
Newton gave us a word for it. Einstein said it bends spacetime. I am saying gravity shortens lifespan.
Read on, and tell me what you think.
I am not saying “gravity exists” or “blood flow matters.” Everyone knows that. The new part is connecting these 3 known facts:
1. Upright posture reduces blood flow to the brain by small amounts, compounding every day for decades.
2. Neurons in the hypothalamus and brainstem are highly metabolic and depend on tiny perforating blood vessels, which makes them very sensitive to even small drops in blood flow.
3. These regions control aging itself: Hormones, inflammation, metabolism, autonomic balance, repair.
Nobody has put these 3 pieces together into a single, testable mechanism for aging. A big part of human aging might be brain-first and gravity driven, and not body-first (like we’ve always believed). That’s the breakthrough.
The more time we spent asking scientists to poke holes in this, the more I began to feel like we were tracing the outline of something hidden in plain sight.
Clues kept appearing. Scattered across biology, physics, medicine, and even evolution. The list runs long.
1. Upright posture reduces blood flow to the brain by small amounts, compounding every day for decades.
2. Neurons in the hypothalamus and brainstem are highly metabolic and depend on tiny perforating blood vessels, which makes them very sensitive to even small drops in blood flow.
3. These regions control aging itself: Hormones, inflammation, metabolism, autonomic balance, repair.
Nobody has put these 3 pieces together into a single, testable mechanism for aging. A big part of human aging might be brain-first and gravity driven, and not body-first (like we’ve always believed). That’s the breakthrough.
The more time we spent asking scientists to poke holes in this, the more I began to feel like we were tracing the outline of something hidden in plain sight.
Clues kept appearing. Scattered across biology, physics, medicine, and even evolution. The list runs long.
So what can we do about this?
We don’t have to escape gravity to counteract the negative effects it has on us. It’s necessary for us to live. Like astronauts, our health would deteriorate in <1G.
We just have to learn how to live longer and better at 1G.
Science already knows that Brain Flow is highly correlated to age, and an important biomarker for aging. However, we don’t pay much attention to Brain Flow.
So first and foremost, all of us need to start recognising how important blood flow to the brain is to our health, and longevity.
We don’t have to escape gravity to counteract the negative effects it has on us. It’s necessary for us to live. Like astronauts, our health would deteriorate in <1G.
We just have to learn how to live longer and better at 1G.
Science already knows that Brain Flow is highly correlated to age, and an important biomarker for aging. However, we don’t pay much attention to Brain Flow.
So first and foremost, all of us need to start recognising how important blood flow to the brain is to our health, and longevity.
TL; DR: ⚠️ CHECKLIST
If you have any of these:
•PLT > 450k
•MPV > 11.5
•D-dimer > 0.50 (especially >1.0)
•Fibrinogen > 400
•CRP > 10
•Ferritin high for sex
•VWF > 150%
•Factor VIII > 150%
Doctors in transplantation medicine have shaped my thinking more than almost any group during this pandemic.. not because they had the answers early on, but because watching their early wrong answers play out has been deeply instructive.
Many dismissed viral persistence.
Many rejected the possibility of blood contamination.
And almost all recoiled at the “airborne AIDS” framing … not because the science wasn’t concerning, but because the analogy made them uncomfortable.
Meanwhile, the data accumulated.
And my stance is now extremely simple:
No one should donate blood without a negative PCR test— for both active and lingering SARS-CoV-2.
And if the recent platelet-sequestration data are confirmed?
Blood donation as we currently practice it will need to stop. Full stop.
Eventually, society will realize that the safest donors are the remaining Novids.
And yes…. at that point the world can cut me a billion-dollar check and I’ll happily “go to work” donating alongside the other viral unicorns.
But let’s stop pretending this is complicated:
If I’m dying and need an organ? I take the organ. The alternative is death.
Blood supply safety? That’s policy. That’s preventable. That’s something we can control.
We learned from HIV.
We built entire safety systems around it.
And yet somehow when it comes to SARS-CoV-2 we’ve learned absolutely nothing.
This will go down as one of the most shocking public-health failures of the century.
Want to check your platelets and related biomarker signals for clotting, etc?
1️⃣ Platelet Count (PLT)
•Normal: 150–450 ×10⁹/L
•High: > 450 ×10⁹/L
→ may indicate reactive thrombocytosis, inflammation, clotting activity.
•Low: < 150 ×10⁹/L
→ can also occur in SARS-CoV-2 from platelet destruction/consumption.
⚠️ Either high or low platelets can signal COVID-related dysregulation.
2️⃣ Mean Platelet Volume (MPV)
•Normal: 7.5–11.5 fL
•High: > 11.5 fL
→ platelets are larger, younger, and more reactive → pro-clotting state.
3️⃣ D-Dimer (DDI)
•Normal: < 0.50 μg/mL FEU (varies slightly by lab)
•High: > 0.50 μg/mL
→ indicates active clot formation + breakdown.
•Very high (COVID-typical): > 1.0–2.0 μg/mL
→ seen in acute COVID and in persistent viral/blood involvement.
4️⃣ Fibrinogen (FIB)
•Normal: 200–400 mg/dL
•High: > 400 mg/dL
→ inflammatory, pro-thrombotic state.
•Very high: > 600 mg/dL
→ common in COVID-related clotting cascades.
5️⃣ CRP or hs-CRP (C-Reactive Protein)
•Normal:
•CRP: < 10 mg/L
•hs-CRP: < 3 mg/L
•High CRP: > 10 mg/L
→ systemic inflammation.
•Very high: > 20–30 mg/L
→ often seen in post-COVID endothelial injury.
6️⃣ Ferritin (FER)
•Normal:
•Men: 30–400 ng/mL
•Women: 15–150 ng/mL
•High:
•Men: > 400 ng/mL
•Women: > 150–200 ng/mL
⚠️ High ferritin is an inflammatory clotting co-marker in COVID.
7️⃣ Von Willebrand Factor (VWF)
•Normal: 50–150%
•High: > 150%
→ endothelial injury; elevated for months post-COVID.
•Very high: > 200%
→ strong indicator of endothelial activation / damage.
8️⃣ Factor VIII (FVIII)
•Normal: 50–150%
•High: > 150%
→ hypercoagulable.
•Very high: > 200%
→ strongly associated with COVID-linked clotting.
9️⃣ Platelet Activation Markers (not routine but important)
If measured:
Soluble P-selectin (sP-sel)
•High: > 40–45 ng/mL
→ platelet activation.
CD62P / CD63 (flow cytometry)
•High expression → activated platelets = higher clot risk.
🔟 aPTT & PT/INR
These are often normal in COVID-related microclotting.
But if abnormal:
aPTT (Intrinsic pathway)
•Normal: 25–35 sec
•Low (fast): < 25 → hypercoagulable
PT/INR (Extrinsic pathway)
•Normal INR: 0.9–1.1
•Low: < 0.9 → hypercoagulable
If you have any of these:
•PLT > 450k
•MPV > 11.5
•D-dimer > 0.50 (especially >1.0)
•Fibrinogen > 400
•CRP > 10
•Ferritin high for sex
•VWF > 150%
•Factor VIII > 150%
Doctors in transplantation medicine have shaped my thinking more than almost any group during this pandemic.. not because they had the answers early on, but because watching their early wrong answers play out has been deeply instructive.
Many dismissed viral persistence.
Many rejected the possibility of blood contamination.
And almost all recoiled at the “airborne AIDS” framing … not because the science wasn’t concerning, but because the analogy made them uncomfortable.
Meanwhile, the data accumulated.
And my stance is now extremely simple:
No one should donate blood without a negative PCR test— for both active and lingering SARS-CoV-2.
And if the recent platelet-sequestration data are confirmed?
Blood donation as we currently practice it will need to stop. Full stop.
Eventually, society will realize that the safest donors are the remaining Novids.
And yes…. at that point the world can cut me a billion-dollar check and I’ll happily “go to work” donating alongside the other viral unicorns.
But let’s stop pretending this is complicated:
If I’m dying and need an organ? I take the organ. The alternative is death.
Blood supply safety? That’s policy. That’s preventable. That’s something we can control.
We learned from HIV.
We built entire safety systems around it.
And yet somehow when it comes to SARS-CoV-2 we’ve learned absolutely nothing.
This will go down as one of the most shocking public-health failures of the century.
Want to check your platelets and related biomarker signals for clotting, etc?
1️⃣ Platelet Count (PLT)
•Normal: 150–450 ×10⁹/L
•High: > 450 ×10⁹/L
→ may indicate reactive thrombocytosis, inflammation, clotting activity.
•Low: < 150 ×10⁹/L
→ can also occur in SARS-CoV-2 from platelet destruction/consumption.
⚠️ Either high or low platelets can signal COVID-related dysregulation.
2️⃣ Mean Platelet Volume (MPV)
•Normal: 7.5–11.5 fL
•High: > 11.5 fL
→ platelets are larger, younger, and more reactive → pro-clotting state.
3️⃣ D-Dimer (DDI)
•Normal: < 0.50 μg/mL FEU (varies slightly by lab)
•High: > 0.50 μg/mL
→ indicates active clot formation + breakdown.
•Very high (COVID-typical): > 1.0–2.0 μg/mL
→ seen in acute COVID and in persistent viral/blood involvement.
4️⃣ Fibrinogen (FIB)
•Normal: 200–400 mg/dL
•High: > 400 mg/dL
→ inflammatory, pro-thrombotic state.
•Very high: > 600 mg/dL
→ common in COVID-related clotting cascades.
5️⃣ CRP or hs-CRP (C-Reactive Protein)
•Normal:
•CRP: < 10 mg/L
•hs-CRP: < 3 mg/L
•High CRP: > 10 mg/L
→ systemic inflammation.
•Very high: > 20–30 mg/L
→ often seen in post-COVID endothelial injury.
6️⃣ Ferritin (FER)
•Normal:
•Men: 30–400 ng/mL
•Women: 15–150 ng/mL
•High:
•Men: > 400 ng/mL
•Women: > 150–200 ng/mL
⚠️ High ferritin is an inflammatory clotting co-marker in COVID.
7️⃣ Von Willebrand Factor (VWF)
•Normal: 50–150%
•High: > 150%
→ endothelial injury; elevated for months post-COVID.
•Very high: > 200%
→ strong indicator of endothelial activation / damage.
8️⃣ Factor VIII (FVIII)
•Normal: 50–150%
•High: > 150%
→ hypercoagulable.
•Very high: > 200%
→ strongly associated with COVID-linked clotting.
9️⃣ Platelet Activation Markers (not routine but important)
If measured:
Soluble P-selectin (sP-sel)
•High: > 40–45 ng/mL
→ platelet activation.
CD62P / CD63 (flow cytometry)
•High expression → activated platelets = higher clot risk.
🔟 aPTT & PT/INR
These are often normal in COVID-related microclotting.
But if abnormal:
aPTT (Intrinsic pathway)
•Normal: 25–35 sec
•Low (fast): < 25 → hypercoagulable
PT/INR (Extrinsic pathway)
•Normal INR: 0.9–1.1
•Low: < 0.9 → hypercoagulable
FYI
I'm about to turn 46.
Here are 33 micro-habits that have quietly changed my life:
1. Drinking water before coffee. When we wake up, we naturally produce cortisol. We are also dehydrated. We don't need to mainline caffeine. We need water.
Here are 33 micro-habits that have quietly changed my life:
1. Drinking water before coffee. When we wake up, we naturally produce cortisol. We are also dehydrated. We don't need to mainline caffeine. We need water.
2. Brushing + flossing my teeth in the shower. I have trouble with flossing, so combining both makes it more likely to happen.
3. Wearing blue light blockers at night. This helps me block blue light, which in turn improves the quality of my sleep.
3. Wearing blue light blockers at night. This helps me block blue light, which in turn improves the quality of my sleep.
4. Walking using an under-the-desk treadmill. This helps me get my steps in while I'm in meetings or doing work.
5. Before I leave my desk for the day, I raise my table and put the treadmill underneath. This forces me to take steps in the morning.
5. Before I leave my desk for the day, I raise my table and put the treadmill underneath. This forces me to take steps in the morning.
Generación Z, Millennials y Boomers inician marcha en el Ángel de la Independencia.
📹 @BiciManager
📹 @BiciManager
Ante la poca afluencia de jóvenes a la marcha identificada como Generación Z, el señor Rodrigo afirma que “el narcogobierno ha sido eficaz en domesticar a los jóvenes".
📹 @BiciManager
📹 @BiciManager
@BiciManager La marcha de jóvenes que se identifican con el movimiento “Generación Z” inició este sábado 15 de noviembre y avanza del Ángel de la Independencia hacia el Centro Histórico de la Ciudad de México.
lasillarota.com/metropoli/2025…
lasillarota.com/metropoli/2025…
Survival Briefing — Nov 15, 2025
If you want to understand what’s actually happening in this country right now, start here:
The most dangerous files in America are the Epstein files.
Not because of what’s trending in Epstein news.
But because the documented chain-of-custody shows who has been holding them all this time, who shaped them, and who is still controlling what the public gets to see.
A thread because this is the TOJ and no I won’t shut up 🖤
If you want to understand what’s actually happening in this country right now, start here:
The most dangerous files in America are the Epstein files.
Not because of what’s trending in Epstein news.
But because the documented chain-of-custody shows who has been holding them all this time, who shaped them, and who is still controlling what the public gets to see.
A thread because this is the TOJ and no I won’t shut up 🖤
2/
Jeffrey Epstein dies August 2019.
His entire digital and paper archive passes to his estate.
The two co-executors who have had sole control of every email, ledger, contact book, and safe since that day are:
Darren Indyke – Epstein’s personal lawyer for 25 years
Richard Kahn – Epstein’s accountant for over 20 years
Both men have been sued by victims for allegedly building and running the financial infrastructure of the trafficking network.
Both men show up in his emails, as trusted placements.
Both men still control the narrative.
Jeffrey Epstein dies August 2019.
His entire digital and paper archive passes to his estate.
The two co-executors who have had sole control of every email, ledger, contact book, and safe since that day are:
Darren Indyke – Epstein’s personal lawyer for 25 years
Richard Kahn – Epstein’s accountant for over 20 years
Both men have been sued by victims for allegedly building and running the financial infrastructure of the trafficking network.
Both men show up in his emails, as trusted placements.
Both men still control the narrative.
3/
August 2025: House Oversight subpoenas the estate.
Indyke and Kahn decide what gets turned over and when.
They do “rolling production.” Translation: they drip-feed
Of these documents on their schedule.
No independent forensic team ever images the servers. 🙃
No third-party custodian.
Just two accused enablers deciding what the American people get to see.
That alone should make your instincts scream.
August 2025: House Oversight subpoenas the estate.
Indyke and Kahn decide what gets turned over and when.
They do “rolling production.” Translation: they drip-feed
Of these documents on their schedule.
No independent forensic team ever images the servers. 🙃
No third-party custodian.
Just two accused enablers deciding what the American people get to see.
That alone should make your instincts scream.
🚨BREAKING: Three electricity transmission towers in Venezuela's Juan Antonio Sotillo municipality, Anzoátegui state, were attacked and toppled.
The country's National Electric Corporation, Corpoelec, has described it as sabotage, leaving a significant part of the local population without power.
The country's National Electric Corporation, Corpoelec, has described it as sabotage, leaving a significant part of the local population without power.
🚨UPDATE: National Electric Corporation of Venezuela, Corpoelec: "This deliberate act seeks to destabilize an essential public service and disrupt the peace of the people in the eastern region of the country. Specialized personnel are already carrying out repair work on the compromised infrastructure.”
Tired of the hollow hum of engagement farming on X? Many large influencer accounts are “controlled opposition,” penning tired legends, their tales remixed and spiral like frayed threads from a loom long abandoned. Their words shimmer false upon the stone— a brittle facade of
truth crumbling under scrutiny—not born of their own breath, but shaped by the unseen hands of power’s puppeteers. These voices are but ghosts, hollowed vessels channeling a script etched in shadow, their every syllable a puppet’s dance beneath the strings. Here, amid the echoes
and the chaos— I carve fractures in the veil, offering whispers untainted, a ripple of raw clarity. I am @CarliFrueh —beckoning you into the anomaly’s embrace—what truth will you forge beyond the illusion?
TLにあったのを何気に読んで、この方及びコメント欄に漂う独特の「戦い」vibeに圧倒された。
男vs女
過去現在未来
復讐の衝動
執念
表明
私が常々「憎み続けるとか恨み続ける感情」は、自分の中にはないなぁと言っている種類の感情だから、興味深く読んだ。
殺害予告とかと同じ種類というか、(続
男vs女
過去現在未来
復讐の衝動
執念
表明
私が常々「憎み続けるとか恨み続ける感情」は、自分の中にはないなぁと言っている種類の感情だから、興味深く読んだ。
殺害予告とかと同じ種類というか、(続
続)むしろ堂々と表明していて、でもこの場合は加害側の名を晒していないか。
でもフォロワー数とバズりの力でSNS上では外野による本人特定もできちゃう可能性もある中、本気で復讐する気があるんだなぁと伝わって来る。
ご年齢から中学生時代は、四半世紀以上前のことになる。ご本人が作家として(続
でもフォロワー数とバズりの力でSNS上では外野による本人特定もできちゃう可能性もある中、本気で復讐する気があるんだなぁと伝わって来る。
ご年齢から中学生時代は、四半世紀以上前のことになる。ご本人が作家として(続
続)成功し名声を得ていても尚、そういう記憶は昇華されるどころか、より力強く燃えたぎるんだなぁ...これ相手が事故で死んだとか、もしくは路上生活をしているとか、何かしら不遇な現在や結果を知ったら、ざまあみろ因果応報だ、とスッキリするのかなぁ。
美容外科医って肩書だけで、なんか荒稼ぎ(続
美容外科医って肩書だけで、なんか荒稼ぎ(続