Let this sink in—All 6 high school basketball 🏀 players in this photo tested positive for #COVID19 in last 4 day… Saturday, Sunday, Monday, Tuesday. None of them wore a mask in any legit way. 1 in 7 infected kids get #LongCovid—➡️4 out of 30-kid COVID+ class.
📊SEVERITY IN KIDS—To debunk/educate those asking if #Omicron is more severe versus Delta in kids relative to adults—let’s look at new official 🇿🇦CDC’s data—➡️case-hospitalization rate is HIGHER now in kids vs Delta, & higher than adults! (Look at chart below—even risk adjusted).
2) Don’t believe South Africa 🇿🇦 CDC’s official report? Want to see US data showing similarly phenomena? Utah also shows troubling trends in severity in youngest kids surpassing per capita hospitalization in middle age adults—(Not vaccine adjusted like SA is above, but still bad)
3) But but but is “case-hospital admission rates” indicative of severity?? Let’s goto the South Africa 🇿🇦 @nicd_sa report’s language directly… “case admission rates are indicative of the severity of the infection—➡️ higher case admission rate indicates more severe infections” 👀
⚠️SUB-VARIANT SURGING—Omicron 2.0 is rising—the #BA2 sublineage of #Omicron has *tripled* in one week, dislodging the old Omicron from total dominance—BA2 now at 3.6% and climbing fast. Expect to be dominant by March. Omicron reinfections are possible. 🧵 covid.cdc.gov/covid-data-tra…
2) Omicron subvariant #BA2 now seems to be surging nationwide (pink), especially in the coastal regions of the Us the fastest. This means it’s not a fluke increase. US’s #BA2 trend will follow Europe’s rise for sure.
3) I’ve been worried about #BA2 for a while. In 3rd week of January, it was rising in Denmark— it’s now near total dominant in Denmark.
🧬Wow—REAWAKENING ancient virus remnants in human DNA—new study finds #SARSCoV2 “can induce (dormant) HERV-W ENV expression” in T-cells & 🧠 cells of those with symptomatic/severe #COVID19” & ➡️ may cause “pathogenic features underlying acute and post-acute COVID”. 📌Avoid COVID!
2) “SARS-CoV-2 can induce HERV-W ENV expression in cells from individuals with symptomatic and severe COVID-19. ➡️ Our data suggest that HERV-W ENV is likely to be involved in pathogenic features underlying symptoms of acute and post-acute COVID.” 👀 medrxiv.org/content/10.110…
3) Look, it’s like this — COVID somehow reactivates a dormant HERV-W protein coded in our DNA that originally is from an ancient virus that had integrated with our DNA. But the coronavirus infection reactivates it’s somehow — and then in turn may make COVID even more severe! Bad!
RIP Dr Li Wenliang who first sounded the alarm🚨 in Wuhan—but muzzled by his hospital & leaders when he tried to warn. Authorities didn’t listen. He shouted over & over—almost terminated from his job for trying to warn the 🌍. Until it was too late. ➡️ We still have not learned!
2) Dr Li was reprimanded for “spreading falsehoods” about the virus. The government later posthumously rescinded the reprimand after his death. Too little too late. His wife was pregnant with their 2nd child when he died. She gave birth in June as a widow. npr.org/sections/coron…
3) Many mourned and paid tribute to Dr Li Wenliang after his death. Like countless others, I couldn’t stop crying in February 2020 when he died. Let’s please not let him die in vain!!! 🙏
NEW—White-tailed deer on NY Staten Island have been found carrying the #Omicron variant—marking the first time Omicron has been reported in wild animals. The results intensify concerns that deer could become a reservoir & a potential source of new variants nytimes.com/2022/02/07/hea…
2) The researchers also found that one deer with Omicron already had a high level of antibodies, suggesting that it may have been previously infected. If #Omicron is similarly immune-evasive in deer, animals infected during earlier outbreaks may be vulnerable to reinfection.
3) “longer-term, widespread circulation of the virus in deer would give the virus more opportunity to mutate, potentially giving rise to new variants that could spill over into people. “The circulation of the virus in deer provides opportunities for it to adapt and evolve,”
📍“Cryptic” unknown #SARSCoV2 variants were found in New York City sewers, many never before found in humans. Some could infect cells with mouse🐁 and/or rat🐀 ACE2-receptor, & were resistant to different neutralizing monoclonal antibodies. So 🍕rat-COVID? nature.com/articles/s4146…
2) “Interestingly, all four WNY lineages contained a polymorphism at spike protein residue 498 (Q498H or Q498Y)… both of these polymorphisms have been associated with host range expansion of SARS-CoV-2 into rodents, which are generally resistant to the parent SARS-CoV-2 lineage”
3) It’s very clear that the 4 key NYC sewer #SARSCoV2 variants have an unusual affinity towards favoring mice-ACE2 and rat-ACE2, much higher than human-ACE2 receptor. Hence the reasons the authors think it has mice 🐁 and rat 🐀 as a likely source.