, 8 tweets, 3 min read
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1/ I’d like to address @JTMarlin77’s point in depth...

For what it’s worth, I think it’s not only fair to critize my work, it’s something I invite. Let’s debate the theories, methodologies, and data...
2/ In fact, I proactively seek out educated, thoughtful opinions that oppose my hypotheses. I don’t just retweet these individuals, I invite them onto the channel (#CholesterolScience series) and even have amazing people like @DrNadolsky write us a pro LDL lowing case for CC.
3/ I bring this up because — like these same people — I often point to associations out loud all the time.

An example would be the thread @JTMarlin77 was referring to where I pointed out my surprising reduction in symptomatic bacterial / viral sickness since my LDL increased.
4/ I think his concern is that this would be taken as an equivalent to my saying, “high LDL 100% causally reduces your chances of getting sick, no question.”

I can express that’s not what I said, but there are people who might simplify it to that anyway.
5/ But to be sure, that’s a risk with *everyone*, including the same critics of my discussions.

I don’t know how anyone can have a sizable following yet won’t have their statements/nuance oversimplified by some.
6/ In spite of this, I further put a lot of effort into emphasizing associations and correlations as opposed to cause and causation where the latter isn’t established. (Which I wish were more common for each side of a debate)
7/ Moreover, I’m human, and have a long history of even tweeting when I’ve misspoke or have been wrong about something, which has happened a number of times. (Check previous tweets for examples)
8/ All I ask is that if you would like to hold me to a standard, that you clearly hold everyone else to the same standard of potentially being misinterpreted by others.
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