What a fear-mongering headline. The treatments didn't cause genetic changes; they caused changes in the way the genes are expressed in the brain. This isn't too surprising given that the 2 soy diets (high PUFA; high oleic) significantly ⬆️weight, worsened glucose tolerance.
it's also been known for quite a while that coconut oil high fat diets don't induce MetS in rodents. There is something weird about them. this was a major area of exploration for my dissertation work ( clinicaltrials.gov/ct2/show/NCT03…). Headline cld easily be "Coconut Oil is Special"
can go back to 2006 and find data that CO and high fish oil diets don't do what high lard and high olive oil diets do for inducing insulin resistance in rats.
ncbi.nlm.nih.gov/pubmed/16720718
ncbi.nlm.nih.gov/pubmed/16720718
Also evidence in the lit that Oleic Acid isn't that great either, even relative to palmitate:
sciencedirect.com/science/articl…
sciencedirect.com/science/articl…
the relevance of these sorts of modified fat composition high fat feeding studies to human nutrition is....extremely limited on their own. many of them don't pan out in human studies, mostly cause folks aren't overeating them constantly like rodents do.
we get these sort of headlines all of the time though: 
part of what makes me doubt them? the high linoleic acid effect is blunted by adding in preformed omega 3s. it reveals an underlying biology that might be interesting about how to perturb feeding behavior,reveal pathways like cannabinoid signaling/oxytocin ncbi.nlm.nih.gov/pubmed/22334255
but n3 metabolism is quite different in mice. and if the addition of lcn3pufa is enough to blunt the obesogenic effect in mice, yet in human studies where folks are given prescription n3s we see no budge in weight status over years of supplementation...well what does that tell u?
