1/ Thank you, Danny — this is a considerably important topic for me, of course.
“Atherogenic dyslipidemia” (AD) is already commonly addressed in the literature and defined as having low HDL and high triglycerides (along with a high preponderance of small dense LDL particles)...
“Atherogenic dyslipidemia” (AD) is already commonly addressed in the literature and defined as having low HDL and high triglycerides (along with a high preponderance of small dense LDL particles)...
2/ In a sense, the “low carb lipid triad” (⬆️LDLc+⬆️HDLc+⬇️TG) I’m interested in is nearly the opposite of AD.
This is especially relevant to the topic — as I hypothesize when ApoB is matched with ⬆️HDLc+⬇️TG, we will frequently see it showing low rates of cardiovascular disease
This is especially relevant to the topic — as I hypothesize when ApoB is matched with ⬆️HDLc+⬇️TG, we will frequently see it showing low rates of cardiovascular disease
3/ This is part of the problem I have where looking at either non-HDL or ApoB —> both lump remnants together with LDLc/p
If we know atherogenic dyslipidemia with low levels of LDLc/p has a high rate of atherosclerosis, why not be interested in the reverse as well?
If we know atherogenic dyslipidemia with low levels of LDLc/p has a high rate of atherosclerosis, why not be interested in the reverse as well?
4/ Like many of my hypotheses, I’m making this before I have the data in hand. But yes, I’d be willing to bet datasets that include ApoB will show this pattern:
Low CVD where ⬆️ApoB+⬆️HDL+⬇️TG
Low CVD where ⬆️ApoB+⬆️HDL+⬇️TG
